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Congmin Wang

Researcher at Fudan University

Publications -  7
Citations -  711

Congmin Wang is an academic researcher from Fudan University. The author has contributed to research in topics: Subventricular zone & Neuroblast. The author has an hindex of 6, co-authored 7 publications receiving 651 citations. Previous affiliations of Congmin Wang include Hebei University of Engineering.

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Identification and characterization of neuroblasts in the subventricular zone and rostral migratory stream of the adult human brain

TL;DR: Evidence that neuroblasts exist continuously in the anterior ventral SVZ and RMS of the adult human brain is provided and the data suggest that the SVZ maintains the ability to produce neuroblast in the adulthuman brain.
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Subcortical origins of human and monkey neocortical interneurons

TL;DR: It is proposed that the majority of primate neocortical GABAergic interneurons originate from ganglionic eminences of the ventral telencephalon, and this work reveals that the mammalian neocortex shares basic rules for interneuron development, substantially reshaping the understanding of the origin and classification ofPrimate neocortex.
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Human and Monkey Striatal Interneurons Are Derived from the Medial Ganglionic Eminence But Not from the Adult Subventricular Zone

TL;DR: Although a small number of neuroblasts exist in the adult human SVZ, they do not migrate into the striatum and become mature striatal interneurons, providing strong evidence that the vast majority of interneuron are generated from the medial ganglionic eminence during embryonic developmental stages, as they are in rodents.
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Nuclear receptor COUP‐TFII‐expressing neocortical interneurons are derived from the medial and lateral/caudal ganglionic eminence and define specific subsets of mature interneurons

TL;DR: It is demonstrated that the POA and ventral MGE do not give rise to COUP‐TFII+ neocortical interneurons, suggesting that the dMGE is the source of COUP'SOM+/somatostatin+/SOM+, which are mainly located in layer V of the adult neocortex.
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Emx1-expressing neural stem cells in the subventricular zone give rise to new interneurons in the ischemic injured striatum.

TL;DR: Genetic fate mapping reveals that newly born CR+ interneurons are generated from Emx1‐expressing neural stem cells in the dorsal–lateral SVZ, suggesting that the fate of the EmX1‐ expressing lineage in the ischemic damaged striatum is restricted.