F
Feriha Ercan
Researcher at Marmara University
Publications - 239
Citations - 5474
Feriha Ercan is an academic researcher from Marmara University. The author has contributed to research in topics: Malondialdehyde & Myeloperoxidase. The author has an hindex of 38, co-authored 214 publications receiving 4830 citations. Previous affiliations of Feriha Ercan include Akdeniz University.
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Simvastatin attenuates cisplatin-induced kidney and liver damage in rats.
TL;DR: Simvastatin is beneficial in cisplatin-induced kidney and liver dysfunction and organ damage in rats via prevention of lipid peroxidation and tissue fibrosis, preservation of antioxidant glutathione, and suppression of neutrophil infiltration.
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β-glucan ameliorates methotrexate-induced oxidative organ injury via its antioxidant and immunomodulatory effects
TL;DR: The findings of the present study suggest that beta-glucan, through its antioxidant and immunoregulatory effects, may be of therapeutic value in alleviating the leukocyte apoptosis, oxidative tissue injury and thereby the intestinal and hepatorenal side effects of methotrexate treatment.
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Taurine protects against methotrexate-induced toxicity and inhibits leukocyte death.
Mustafa Çetiner,Göksel Şener,A. Özer Şehirli,Emel Eksioglu-Demiralp,Feriha Ercan,Serap Sirvanci,Nursal Gedik,Sertac Akpulat,Tulay Tecimer,Berrak Ç. Yeğen +9 more
TL;DR: It appears that taurine protects against methotrexate-induced oxidant organ injury and inhibits leukocyte apoptosis and may be of therapeutic potential in alleviating the systemic side effects of chemotherapeutics.
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Protective effect of β-glucan against oxidative organ injury in a rat model of sepsis
TL;DR: It seems likely that beta-glucan protects against sepsis-induced oxidative organ injury, as it significantly decreased GSH levels and increased the MDA levels and MPO activity and elevated plasma TNF-alpha levels in septic rats, causing oxidative damage.
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Oxytocin protects against sepsis-induced multiple organ damage: role of neutrophils.
TL;DR: OT protects against sepsis-induced oxidative damage by acting as an antioxidant agent and its protective effect in the colon and liver appears to be dependent on its inhibitory effect on neutrophil infiltration.