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Fernando dos Santos

Researcher at University of São Paulo

Publications -  39
Citations -  380

Fernando dos Santos is an academic researcher from University of São Paulo. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 11, co-authored 29 publications receiving 283 citations.

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Journal ArticleDOI

Hypertension, Blood Pressure Variability, and Target Organ Lesion.

TL;DR: Evidence relating BPV and target organ damage in hypertension in clinical and experimental settings is presented and some evidence implicated in increased CV risk in hypertension is presented.
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Preventive role of exercise training in autonomic, hemodynamic, and metabolic parameters in rats under high risk of metabolic syndrome development

TL;DR: Correlation analysis showed that both Lee index and kITT were associated with vagal impairment caused by fructose, and exercise training plays a preventive role in both autonomic and hemodynamic alterations related to the excessive fructose consumption.
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Generalization of feature embeddings transferred from different video anomaly detection domains

TL;DR: This paper reports a study on video anomaly detection, focusing on the analysis of feature embeddings of pre-trained CNNs with the use of novel cross-domain generalization measures that allow to study how source features generalize for different target video domains.
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Cardiac dysfunction in Pkd1-deficient mice with phenotype rescue by galectin-3 knockout.

TL;DR: Myocardial dysfunction occurs in different Pkd1-deficient models and suppression of galectin-3 expression rescues this phenotype, and analysis of a severe cystic model showed that PkD1(V/V);Lgals3(-/-) (VVG-) mice have longer survival, decreased cardiac apoptosis and improved heart function compared to VVG+.
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The role of the baroreflex and parasympathetic nervous system in fructose-induced cardiac and metabolic alterations

TL;DR: Chronic fructose overload induced severe baroreflex impairment, and the compensatory enhancement in parasympathetic function in SAD rats submitted to fructose intake may point out the possibility of use of approaches that improve vagal function as therapeutic target to attenuate fructose-induced cardiometabolic dysfunctions.