F
François Rieger
Researcher at French Institute of Health and Medical Research
Publications - 136
Citations - 3851
François Rieger is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Skeletal muscle & Acetylcholinesterase. The author has an hindex of 32, co-authored 136 publications receiving 3784 citations. Previous affiliations of François Rieger include Boston Children's Hospital & Centre national de la recherche scientifique.
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Altered expression of neuronal cell adhesion molecules induced by nerve injury and repair.
TL;DR: The data suggest that local signals between neurons and glia may regulate CAM expression in the spinal cord and nerve during regeneration, and that activity may regulate N-CAM expression in muscle.
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N-CAM at the vertebrate neuromuscular junction.
TL;DR: The findings indicate that, in muscle, in addition to chemical modulation, cell-surface modulation of N-CAM occurs both in amount and distribution during embryogenesis and in response to denervation.
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N-cadherin and N-CAM in myoblast fusion: compared localisation and effect of blockade by peptides and antibodies
TL;DR: Observations suggest that N-cadherin, acting independently from N-CAM, is a major cell adhesion molecule involved in embryonic myoblast fusion in vitro.
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Neuronal Cell Adhesion Molecules and Cytotactin Are Colocalized at the Node of Ranvier
François Rieger,Joanne K. Daniloff,Martine Pinçon-Raymond,Kathryn L. Crossin,Martin Grumet,Gerald M. Edelman +5 more
TL;DR: Findings are consistent with the hypothesis that surface modulation of neuronal CAMs mediated by signals shared between neurons and glia may be necessary for establishing and maintaining the nodes of Ranvier.
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Acetylcholinesterase and butyrylcholinesterase in frontal cortex and cerebrospinal fluid of demented and non-demented patients with Parkinson's disease
TL;DR: Results indicated that AChE and BChE levels were significantly higher in demented parkinsonian patients compared to the non-demented subjects, suggesting that the enzymes were affected in the cholinergic neurones before transport to cortical nerve terminals.