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Frederick C. de Beer

Researcher at University of Kentucky

Publications -  44
Citations -  2996

Frederick C. de Beer is an academic researcher from University of Kentucky. The author has contributed to research in topics: Serum amyloid A & Apolipoprotein B. The author has an hindex of 27, co-authored 44 publications receiving 2691 citations. Previous affiliations of Frederick C. de Beer include Veterans Health Administration.

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Role of Group II Secretory Phospholipase A2 in Atherosclerosis : 1. Increased Atherogenesis and Altered Lipoproteins in Transgenic Mice Expressing Group IIa Phospholipase A2

TL;DR: Data indicate that group IIa sPLA2 may promote atherogenesis, in part, through its effects on lipoprotein levels, and provide a possible mechanism for the observation that there is an increased incidence of coronary artery disease in many chronic inflammatory diseases.
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SR-BII, an isoform of the scavenger receptor BI containing an alternate cytoplasmic tail, mediates lipid transfer between high density lipoprotein and cells.

TL;DR: These studies show that SR-BII, an HDL receptor isoform containing a distinctly different cytoplasmic tail, mediates selective lipid transfer between HDL and cells, but with a lower efficiency than the previously characterized variant.
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Overexpression of Secretory Phospholipase A2 Causes Rapid Catabolism and Altered Tissue Uptake of High Density Lipoprotein Cholesteryl Ester and Apolipoprotein A-I

TL;DR: It is demonstrated that overexpression of sPLA2 alone in the absence of inflammation causes profound alterations of HDL metabolism in vivo and is consistent with the hypothesis that s PLA2 may promote HDL catabolism in acute and chronic inflammatory conditions.
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SR-BI protects against endotoxemia in mice through its roles in glucocorticoid production and hepatic clearance

TL;DR: It is concluded that scavenger receptor B-I in mice is required for the antiinflammatory response to LPS-induced endotoxic shock, likely through its essential role in facilitating glucocorticoid production and LPS hepatic clearance.