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G

G. Leori

Researcher at University of Sassari

Publications -  9
Citations -  672

G. Leori is an academic researcher from University of Sassari. The author has contributed to research in topics: Salmonella enterica & Virulence. The author has an hindex of 8, co-authored 9 publications receiving 627 citations.

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Host adapted serotypes of Salmonella enterica.

TL;DR: It is hoped that an appreciation of the differences that exist in the way these host-adapted serotypes of Salmonella interact with the host will lead to a greater understanding of the complex host–parasite relationship that characterizes salmonella infections.
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Salmonella enterica Serovar-Host Specificity Does Not Correlate with the Magnitude of Intestinal Invasion in Sheep

TL;DR: It is suggested that Salmonella serovar specificity in sheep correlates with bacterial persistence at systemic sites following intravenous inoculation and that theSalmonella pathogenicity island 1 influences but the virulence plasmid genes do not influence the ability of serovars Abortusovis to invade the intestinal mucosa in sheep.
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Strain typing with IS200 fingerprints in Salmonella abortusovis.

TL;DR: Strain typing with IS200 fingerprints proved to be more reliable than plasmid analysis, because the latter yielded a high degree of polymorphism, even among isolates from the same flock.
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Phenotypic features and molecular characterization of plasmids in Salmonella abortusovis

TL;DR: The phenotypic and genotypic characteristics of 69 wild-type strains of Salmonella abortusovis from Sardinia and other Italian regions, representing four different epidemic outbreaks, were studied and allowed us to outline a genetic correlation among S. abortuovis isolated from different outbreaks.
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TnphoA Salmonella abortusovis mutants unable to adhere to epithelial cells and with reduced virulence in mice.

TL;DR: A bank of strains was created in order to identify any phenotypic modification which could affect the periplasmic and/or exported proteins involved in virulence of S. abortusovis TnphoA mutants, since a group of mutant strains has shown reduced virulence in mice and one mutant is completely avirulent.