G
Geoffrey N. Woodruff
Researcher at University of Southampton
Publications - 71
Citations - 1683
Geoffrey N. Woodruff is an academic researcher from University of Southampton. The author has contributed to research in topics: Dopamine & Dopamine receptor. The author has an hindex of 25, co-authored 71 publications receiving 1679 citations. Previous affiliations of Geoffrey N. Woodruff include Kyoto University.
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Journal ArticleDOI
The action of substance P on mesencephalic reticular and substantia nigral neurones of the rat.
Robert J. Walker,Robert J. Walker,J. A. Kemp,J. A. Kemp,H. Yajima,K. Kitagawa,K. Kitagawa,Geoffrey N. Woodruff,Geoffrey N. Woodruff +8 more
TL;DR: Evidence is presented for Substance P as a putative excitatory transmitter onto reticular and nigral neurones possibly released from primary sensory afferents and in some cases this excitation was rapid.
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PRESYNAPTIC γ‐AMINOBUTYRIC ACID RECEPTORS IN THE RAT ANOCOCCYGEUS MUSCLE AND THEIR ANTAGONISM BY 5‐AMINOVALERIC ACID
TL;DR: The results suggest that GABAB receptors are present on motor nerve terminals in the rat anococcygeus muscle and that 5‐aminovaleric acid is an antagonist of these receptors.
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Picrotoxin antagonism of γ aminobutyric acid inhibitory responses and synaptic inhibition in the rat substantia nigra
TL;DR: Iontophoretically applied picrotoxin reversibly blocks both of these inhibitory responses to GABA in the substantia nigra of the rat, consistent with the hypothesis that GABA is the transmitter released by the inhibitory striato-nigral pathway.
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Studies on the pharmacology of neurones in the nucleus accumbens of the rat.
TL;DR: The results are consistent with the suggestion that dopamine is an inhibitory transmitter in the nucleus accumbens and in the caudate nucleus and support the hypothesis that the effects of dopamine are mediated by cyclic AMP.
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Studies on the behavioural pharmacology of a cyclic analogue of dopamine following its injection into the brains of conscious rats
TL;DR: It is concluded that the central stimulant action of ADTN is due to an effect on the dopamine receptors in the nucleus accumbens.