G
George J. Miller
Researcher at St Bartholomew's Hospital
Publications - 115
Citations - 9158
George J. Miller is an academic researcher from St Bartholomew's Hospital. The author has contributed to research in topics: Postprandial & Factor VII. The author has an hindex of 51, co-authored 110 publications receiving 8884 citations.
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Journal ArticleDOI
Plasma fibrinogen level and the risk of major cardiovascular diseases and nonvascular mortality: an individual participant meta-analysis.
John Danesh,Sarah Lewington,Simon G. Thompson,Lowe Gdo.,Rory Collins,John B. Kostis,Angela Wilson,Aaron R. Folsom,K. Wu,M. Benderly,Uri Goldbourt,Johann Willeit,Stefan Kiechl,Yarnell Jwg.,P. M. Sweetnam,P. Elwood,Mary Cushman,Bruce M. Psaty,Russell P. Tracy,Anne Tybjærg-Hansen,F. Haverkate,De Maat Mpm.,Fowkes Fgr.,Amanda J Lee,F.B. Smith,Veikko Salomaa,Kennet Harald,V. Rasi,E. Vahtera,P. Jousilahti,Juha Pekkanen,Ralph B. D'Agostino,W B Kannel,Wilson Pwf.,Geoffrey H. Tofler,Carmen Luisa Arocha-Piñango,Alvaro Rodríguez-Larralde,Elena Nagy,Mercedes E Mijares,Raul Espinosa,E Rodriquez-Roa,Elena Ryder,María Diez-Ewald,Gilberto Campos,Virginia Fernández,Enrique Torres,Roberto Marchioli,Franco Valagussa,Annika Rosengren,Lars Wilhelmsen,Georgios Lappas,Henry Eriksson,Peter Cremer,Dorothea Nagel,J D Curb,Beatriz L. Rodriguez,K Yano,Jukka T. Salonen,Kristiina Nyyssönen,Tomi-Pekka Tuomainen,Bo Hedblad,P. Lind,H. Loewel,Wolfgang Koenig,Tom W. Meade,J.A. Cooper,B L De Stavola,C. Knottenbelt,George J. Miller,Kenneth A. Bauer,Robert D. Rosenberg,S. Sato,A. Kitamura,Yoshihiko Naito,Hiroyasu Iso,T. Palosuo,Pierre Ducimetière,Philippe Amouyel,Dominique Arveiler,Alun Evans,Jean Ferrières,Irène Juhan-Vague,Annie Bingham,Helmut Schulte,Gerd Assmann,Bernard Cantin,Benoît Lamarche,Jean-Pierre Després,Gilles R. Dagenais,Hugh Tunstall-Pedoe,Mark Woodward,Yoav Ben-Shlomo +91 more
TL;DR: In this article, a large-scale individual participant meta-analysis was conducted to assess the relationship of fibrinogen levels with risk of major vascular and non-vascular outcomes based on individual participant data.
Journal ArticleDOI
Fibrinolytic activity, clotting factors, and long-term incidence of ischaemic heart disease in the Northwick Park Heart Study
TL;DR: Low FA appears to be a leading determinant of ischaemic heart disease in younger men and methods of enhancing fibrinolytic activity, whether by life-style changes or pharmacologically, should be considered.
Journal ArticleDOI
The interleukin-6 −174 G/C promoter polymorphism is associated with risk of coronary heart disease and systolic blood pressure in healthy men
TL;DR: It is suggested that, at least in part, the effect of the IL-6 -174G>C polymorphism on blood pressure is likely to be operating through inflammatory mechanisms, but the genotype effect on coronary heart disease risk is largely unexplained by its effect onBlood pressure.
Journal ArticleDOI
Relative contribution of variation within the APOC3/A4/A5 gene cluster in determining plasma triglycerides
Philippa J. Talmud,Emma Hawe,S Martin,Michael Olivier,George J. Miller,Edward M. Rubin,Len A. Pennacchio,Steve E. Humphries +7 more
TL;DR: Variation in APOA5 is associated with differences in TGs in healthy men, independent of those previously reported for APOC3, while association between APOA4 and TG reflects linkage disequilibrium with these sites.
Journal ArticleDOI
Cholesteryl Ester Transfer Protein TaqIB Variant, High-Density Lipoprotein Cholesterol Levels, Cardiovascular Risk, and Efficacy of Pravastatin Treatment Individual Patient Meta-Analysis of 13 677 Subjects
S.M. Boekholdt,Frank M. Sacks,Joop Jukema,J. Shepherd,Dilys J. Freeman,Alex D. McMahon,François Cambien,Viviane Nicaud,G.J. de Grooth,P.J. Talmud,S.E. Humphries,George J. Miller,G. Eiriksdottir,Vilmundur Gudnason,Heikki Kauma,Sakari Kakko,Markku J. Savolainen,Marcello Arca,A. Montali,Simin Liu,H.J. Lanz,A. H. Zwinderman,J. A. Kuivenhoven,John J. P. Kastelein +23 more
TL;DR: The CETP TaqIB variant is firmly associated with HDL-C plasma levels and as a result, with the risk of CAD, and does not influence the response to pravastatin therapy.