G
Gérard Barbanel
Researcher at Centre national de la recherche scientifique
Publications - 54
Citations - 1780
Gérard Barbanel is an academic researcher from Centre national de la recherche scientifique. The author has contributed to research in topics: Median eminence & Long-term potentiation. The author has an hindex of 22, co-authored 54 publications receiving 1701 citations. Previous affiliations of Gérard Barbanel include University of Montpellier.
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Journal ArticleDOI
Temporal cascade of plasma level surges in ACTH, corticosterone, and cytokines in endotoxin-challenged rats
Laurent Givalois,J. Dornand,Mourad Mekaouche,M. D. Solier,A. F. Bristow,G. Ixart,P. Siaud,I. Assenmacher,Gérard Barbanel +8 more
TL;DR: The present study was designed to investigate the coupling mechanisms linking the immune and the neuroendocrine corticotropic systems in an integrated defense response triggered by an infectious aggression.
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Neurodevelopmental damage after prenatal infection: role of oxidative stress in the fetal brain.
Fabien Lanté,Johann Meunier,Janique Guiramand,Tangui Maurice,Mélanie Cavalier,Marie-Céleste de Jesus Ferreira,Rose Aimar,Catherine Cohen-Solal,Michel Vignes,Gérard Barbanel +9 more
TL;DR: It is shown that LPS injection during pregnancy in rats, 2 days before delivery, triggered an oxidative stress in the hippocampus of male fetuses, evidenced by a rapid rise in protein carbonylation and by decreases in alpha-tocopherol levels and in the ratio of reduced/oxidized forms of glutathione (GSH/GSSG).
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Late N-acetylcysteine treatment prevents the deficits induced in the offspring of dams exposed to an immune stress during gestation.
Fabien Lanté,Johann Meunier,Janique Guiramand,Marie-Céleste de Jesus Ferreira,Gilles Cambonie,Gilles Cambonie,Rose Aimar,Catherine Cohen-Solal,Tangui Maurice,Michel Vignes,Gérard Barbanel +10 more
TL;DR: The results point to fetal hippocampal GSH as a major target of the detrimental effects of the in utero LPS challenge, suggesting that this drug might be clinically useful even after an immuno‐inflammatory episode.
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Intrahypothalamic infusion of interleukin-1 beta increases the release of corticotropin-releasing hormone (CRH 41) and adrenocorticotropic hormone (ACTH) in free-moving rats bearing a push-pull cannula in the median eminence.
TL;DR: In two simultaneous studies on unanesthetized rats implanted with either an intracerebral cannula adjacent to the paraventricular nucleus of the hypothalamus and an intracarotid cannula, the effect of interleukin-1 beta infused on the release of corticotropin-releasing hormone (CRH) 41 and adrenocorticotropic hormone (ACTH) was explored.
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Physiology of neural pathways affecting CRH secretion.
TL;DR: Present knowledge of the neural components controlling C R H secretion is evaluated, and most of the recent data in this line of research are still based on measurements of the peripheral hormones of the HPA axis-i.e. of ACTH and sometimes only of corticosterone.