G
Gerard C. L. Wong
Researcher at University of California, Los Angeles
Publications - 183
Citations - 10694
Gerard C. L. Wong is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Antimicrobial peptides & Membrane. The author has an hindex of 51, co-authored 173 publications receiving 8831 citations. Previous affiliations of Gerard C. L. Wong include California Institute of Technology & University of California, Berkeley.
Papers
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Heterogeneity in surface sensing produces a division of labor in Pseudomonas aeruginosa populations
Catherine R. Armbruster,Calvin K. Lee,Jessica Parker-Gilham,J. de Anda,Aiguo Xia,Boo Shan Tseng,Lucas R. Hoffman,Fan Jin,Fan Jin,Caroline S. Harwood,Gerard C. L. Wong,Matthew R. Parsek +11 more
TL;DR: This work describes how the Wsp system generates heterogeneity in surface sensing, resulting in two physiologically distinct subpopulations of cells, and shows that this heterogeneity strongly correlates to surface behavior for descendent cells.
Posted ContentDOI
Extended hopanoid loss reduces bacterial motility and surface attachment and leads to heterogeneity in root nodule growth kinetics in a Bradyrhizobium-Aeschynomene symbiosis
TL;DR: This work provides a quantitative reference point for understanding the phenotypic diversity of ineffective symbionts in A. afraspera and identifies specific developmental stages affected by extended hopanoid loss for future mechanistic work.
Journal Article
Interaction of Arginine-Rich Peptides with Model Cell Membranes
Journal Article
Evolution of growth modes for polyelectrolyte bundles
TL;DR: In this article, the kinetics of aggregation and bundle formation of actin are tracked using two different fluorescently labeled populations of F-actin, and it is found that the growth mode of these bundles evolves with time and salt concentration, varying from an initial lateral growth to a longitudinal one at later stages.
Posted ContentDOI
Fine tuning cyclic-di-GMP signaling in Pseudomonas aeruginosa using the type 4 pili alignment complex
TL;DR: A key implication of this work is that more than just regulating signal levels, cells must modulate the dynamic range of cyclic-di-GMP to precisely control the transition to a biofilm lifestyle.