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Gloria Patricia Cardona-Gómez
Researcher at University of Antioquia
Publications - 80
Citations - 3226
Gloria Patricia Cardona-Gómez is an academic researcher from University of Antioquia. The author has contributed to research in topics: Neuroprotection & Ischemia. The author has an hindex of 26, co-authored 78 publications receiving 2738 citations. Previous affiliations of Gloria Patricia Cardona-Gómez include Spanish National Research Council.
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Journal ArticleDOI
Astrocytic modulation of blood brain barrier: perspectives on Parkinson’s disease
Ricardo Cabezas,Marcos Fidel Avila,Janneth Gonzalez,Ramon dos Santos El-Bachá,Eliana Baez,Luis M. Garcia-Segura,Juan Camilo Jurado Coronel,Francisco Capani,Gloria Patricia Cardona-Gómez,George E. Barreto +9 more
TL;DR: The implications of astrocyte functions in the protection of the blood–brain barrier, and in the development of Parkinson’s disease (PD) and related disorders are discussed.
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The flavonoid quercetin ameliorates Alzheimer’s disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer’s disease model mice
Angélica Maria Sabogal-Guáqueta,Juan Ignacio Muñoz-Manco,José R. Ramírez-Pineda,Marisol Lamprea-Rodriguez,Edison Osorio,Gloria Patricia Cardona-Gómez +5 more
TL;DR: It is suggested that quercetin reverses histological hallmarks of AD and protects cognitive and emotional function in aged 3xTg-AD mice.
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The Role of Astrocytes in Neuroprotection after Brain Stroke: Potential in Cell Therapy.
TL;DR: This review analyzes pro-survival responses of astrocytes that would allow their use in cell therapy strategies and makes them excellent candidates as a source of neuroprotection and neurorestoration in tissues affected by ischemia/reperfusion.
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Interactions of estrogens and insulin-like growth factor-I in the brain: implications for neuroprotection
Gloria Patricia Cardona-Gómez,Pablo Mendez,Lydia L. DonCarlos,Iñigo Azcoitia,Luis M. Garcia-Segura +4 more
TL;DR: Both in vitro and in vivo studies have shown that there is an interaction between ERs and IGF-IR in the promotion of neuronal survival and in the response of neural tissue to injury, suggesting that a parallel activation or co-activation of ERs-IR mediates neuroprotection.
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Silencing of CDK5 reduces neurofibrillary tangles in transgenic alzheimer's mice.
Diego Piedrahita,Israel Hernandez,Alejandro López-Tobón,Dmitry Fedorov,Boguslaw Obara,B.S. Manjunath,Ryan L. Boudreau,Beverly L. Davidson,Frank M. LaFerla,Juan Carlos Gallego-Gómez,Kenneth S. Kosik,Gloria Patricia Cardona-Gómez +11 more
TL;DR: Silencing of CDK5 reduces the phosphorylation of tau in primary neuronal cultures and in the brain of wild-type C57BL/6 mice, and the knockdown strongly decreased the number of neurofibrillary tangles in the hippocampi of triple-transgenic mice.