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Hans Verhoef

Researcher at Wageningen University and Research Centre

Publications -  75
Citations -  2372

Hans Verhoef is an academic researcher from Wageningen University and Research Centre. The author has contributed to research in topics: Iron deficiency & Population. The author has an hindex of 24, co-authored 67 publications receiving 2100 citations. Previous affiliations of Hans Verhoef include Medical Research Council & University of London.

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Hepcidin is the major predictor of erythrocyte iron incorporation in anemic African children.

TL;DR: It is suggested that low-cost point-of-care hepcidin assays would aid iron supplementation programs in the developing world after it was concluded that under conditions of competing signals (anemia, iron deficiency, and infection), hePCidin powerfully controls use of dietary iron.
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Decreased plasma leptin concentrations in tuberculosis patients are associated with wasting and inflammation.

TL;DR: The results do not support the concept that weight loss in tuberculosis is caused by enhanced production of leptin, and loss of body fat leads to low plasma leptin concentrations, and prolonged inflammation may further suppress leptin production.
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Serum transferrin receptor concentration indicates increased erythropoiesis in Kenyan children with asymptomatic malaria

TL;DR: The findings are consistent with the notion that malaria-induced hemolysis is accompanied by increased erythropoiesis, and Serum transferrin receptor concentration is not useful for detecting iron deficiency in individuals with malaria.
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Efficacy of iron-fortified whole maize flour on iron status of schoolchildren in Kenya: a randomised controlled trial

TL;DR: Consumption of whole maize flour fortified with NaFeEDTA caused modest, dose-dependent improvements in children's iron status, and was more suitable than electrolytic iron for supplementation of iron in the diet in high-phytate flours.
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Factors associated with stunting in infants aged 5-11 months in the Dodota-Sire District, rural Ethiopia.

TL;DR: The quality and quantity of foods consumed by infants is insufficient to prevent stunting, and it is necessary to increase the nutrient supply to infants by increasing intake and nutrient concentration of breast milk and of supplementary foods they consume, and by providing supplements to infants where appropriate.