H
Harvey B. Pollard
Researcher at Uniformed Services University of the Health Sciences
Publications - 315
Citations - 15467
Harvey B. Pollard is an academic researcher from Uniformed Services University of the Health Sciences. The author has contributed to research in topics: Chromaffin cell & Exocytosis. The author has an hindex of 64, co-authored 307 publications receiving 14679 citations. Previous affiliations of Harvey B. Pollard include National Institutes of Health & Max Planck Society.
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Journal ArticleDOI
Annexins: the problem of assessing the biological role for a gene family of multifunctional calcium- and phospholipid-binding proteins.
Patrick Raynal,Harvey B. Pollard +1 more
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Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum.
TL;DR: It is proposed that a useful strategy for drug discovery for treatment of Alzheimer disease may include screening compounds for their ability to block or otherwise modify A beta P channels.
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Giant multilevel cation channels formed by Alzheimer disease amyloid beta-protein [A beta P-(1-40)] in bilayer membranes
TL;DR: In this article, the Alzheimer disease 40-residue amyloid beta-protein [A beta P-(1-40)] can form cation-selective channels when incorporated into planar lipid bilayers by fusion of liposomes containing the peptide.
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Molecular dissection of nucleolin’s role in growth and cell proliferation: new insights
TL;DR: Nucleolin is a major nucleolar protein of exponentially growing eukaryotic cells, which is directly involved in the regulation of ribosome biogenesis and maturation, and is a target for regulation by proteolysis, methylation, ADP‐ribosylation, and phosphorylation by CKII, cdc2, PKC‐ξ, cyclic AMP‐dependent protein kinase, and ecto‐protein kinase.
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From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations.
Gudio Veit,Radu G. Avramescu,Annette N. Chiang,Scott A. Houck,Zhiwei Cai,Kathryn W. Peters,Jeong S. Hong,Harvey B. Pollard,William B. Guggino,William E. Balch,William R. Skach,Garry R. Cutting,Raymond A. Frizzell,David N. Sheppard,Douglas M. Cyr,Eric J. Sorscher,Jeffrey L. Brodsky,Gergely L. Lukacs +17 more
TL;DR: Establishment of a set of combinatorial categories of the previously defined basic defects in CF alleles will aid the design of even more efficacious therapeutic interventions for CF patients.