H
Heinz Kutzner
Researcher at University of Graz
Publications - 24
Citations - 2929
Heinz Kutzner is an academic researcher from University of Graz. The author has contributed to research in topics: Dermatopathology & Board certification. The author has an hindex of 8, co-authored 24 publications receiving 2748 citations. Previous affiliations of Heinz Kutzner include Medical University of Graz.
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Journal ArticleDOI
Distinct Sets of Genetic Alterations in Melanoma
John A. Curtin,Jane Fridlyand,Toshiro Kageshita,Hetal N. Patel,Klaus J. Busam,Heinz Kutzner,Kwang Hyun Cho,Setsuya Aiba,Eva B. Bröcker,Philip E. LeBoit,Daniel Pinkel,Boris C. Bastian +11 more
TL;DR: The genetic alterations identified in melanoma at different sites and with different levels of sun exposure indicate that there are distinct genetic pathways in the development of melanoma and implicate CDK4 and CCND1 as independent oncogenes in melanomas without mutations in BRAF or N-RAS.
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Cutaneous adult myofibroma: a vascular neoplasm
TL;DR: There is an adult counterpart of infantile myofibromatosis, characterized by solitary lesions that have a predilection for involve the dermis and show no tendency to regression, although they have an entirely benign biological behavior.
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Cutaneous myoepithelioma: an under-recognized cutaneous neoplasm composed of myoepithelial cells.
TL;DR: In this paper, the authors report five additional examples of purely myoepithelial tumors located exclusively in the dermis, which are well-circumscribed dermal lesions composed of fascicles of spindle cells with eosinophilic cytoplasm and ovoid-to spindle-shaped nuclei.
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Pityriasis lichenoides et varioliformis acuta with numerous CD30(+) cells: a variant mimicking lymphomatoid papulosis and other cutaneous lymphomas. A clinicopathologic, immunohistochemical, and molecular biological study of 13 cases.
TL;DR: The CD30+ PLEVA variant described herein and LyP show considerable overlap if one takes into account all known variations of the 2 conditions recognized in recent years, thus suggesting that LyP and PLEva may be much more biologically closely related entities than currently thought or can even occur on a clinicopathologic spectrum.