H
Hendrik Uyttendaele
Researcher at Columbia University
Publications - 13
Citations - 1614
Hendrik Uyttendaele is an academic researcher from Columbia University. The author has contributed to research in topics: Notch signaling pathway & Morphogenesis. The author has an hindex of 10, co-authored 13 publications receiving 1568 citations.
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Journal ArticleDOI
Notch4/int-3, a mammary proto-oncogene, is an endothelial cell-specific mammalian Notch gene
TL;DR: Comparison of the coding potential of the int-3 gene to that of notch4 suggests that loss of the extracellular domain of Notch4 leads to constitutive activation of this murine Notch protein.
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Follicular unit extraction: minimally invasive surgery for hair transplantation.
William R. Rassman,Robert M. Bernstein,Robert E. McClellan,Roy Jones,Eugene Worton,Hendrik Uyttendaele +5 more
TL;DR: FUE is a minimally invasive approach tohair transplantation that obviates the need for a linear donor incision and can serve as an important alternative to traditional hair transplantation in certain patients.
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Exposing the human nude phenotype
Jorge Frank,Claudio Pignata,Andrei A. Panteleyev,David M. Prowse,Howard P. Baden,Lorin Weiner,L. Gaetaniello,Wasim Ahmad,N. Pozzi,P. B. Cserhalmi-Friedman,Vincent M. Aita,Hendrik Uyttendaele,Derek Gordon,Jurg Ott,Janice L. Brissette,Angela M. Christiano +15 more
TL;DR: A genetic analysis is carried out that reveals a human homologue of the nude mouse, the best known of the ‘bald’ mouse phenotypes, the nude mice.
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Notch4 and wnt-1 proteins function to regulate branching morphogenesis of mammary epithelial cells in an opposing fashion
TL;DR: Branching morphogenesis of the mouse mammary epithelial TAC-2 cell line was used as an assay to examine the role of Wnt, HGF, TGF-beta, and the Notch receptors in branching morphogenesis, suggesting that Wnt and Notch signaling may play opposite roles in mammary gland development.
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Expression of an activated Notch4(int-3) oncoprotein disrupts morphogenesis and induces an invasive phenotype in mammary epithelial cells in vitro.
TL;DR: The results suggest that the ability of Notch4(int‐3) to subvert normal epithelial morphogenesis and to promote invasion of the extracellular matrix contributes significantly to its tumorigenic potential.