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Henning Langberg

Researcher at University of Copenhagen

Publications -  245
Citations -  13537

Henning Langberg is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Tendon & Skeletal muscle. The author has an hindex of 60, co-authored 242 publications receiving 11999 citations. Previous affiliations of Henning Langberg include Novozymes & Norwegian Defence Estates Agency.

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Coordinated collagen and muscle protein synthesis in human patella tendon and quadriceps muscle after exercise

TL;DR: There is a rapid increase in collagen synthesis after strenuous exercise in human tendon and muscle, and the similar time course of changes of protein synthetic rates in different cell types supports the idea of coordinated musculotendinous adaptation.
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Monitoring tissue oxygen availability with near infrared spectroscopy (NIRS) in health and disease.

TL;DR: NIRS has been applied to measure oxygenation in a variety of tissues including muscle, brain and connective tissue, and more recently it has been used in the clinical setting to assess circulatory and metabolic abnormalities.
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Type I collagen synthesis and degradation in peritendinous tissue after exercise determined by microdialysis in humans

TL;DR: It is indicated that acute exercise induces increased formation of type I collagen in peritendinous tissue as determined with microdialysis and using dialysate fibre with a very high molecular mass cut‐off, which suggests an adaptation to acute physical loading also in non‐bone‐related collagen in humans.
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The pathogenesis of tendinopathy: balancing the response to loading

TL;DR: Understanding how tendon tissue adapts to mechanical loading will help to unravel the pathogenesis of tendinopathy, which is characterized by pain during activity, localized tenderness upon palpation, swelling and impaired performance.
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Myofibre damage in human skeletal muscle: effects of electrical stimulation versus voluntary contraction

TL;DR: In human muscle, the delayed onset of muscle soreness was not significantly different between the two treatments despite marked differences in intramuscular histological markers, in particular myofibre proteins and satellite cell markers.