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Hilary Koprowski

Researcher at Thomas Jefferson University

Publications -  336
Citations -  25417

Hilary Koprowski is an academic researcher from Thomas Jefferson University. The author has contributed to research in topics: Virus & Antigen. The author has an hindex of 80, co-authored 336 publications receiving 24997 citations. Previous affiliations of Hilary Koprowski include World Health Organization & Niigata University.

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The specificity of rabies virus RNA encapsidation by nucleoprotein.

TL;DR: Investigation of the interaction between rabies virus N and the positive-strand leader RNA transcript revealed that recombinant N binds preferentially to rabiesirus leader RNA and that N binding to leader RNA was 5 to 10 times stronger than to nonleader RNA.
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Lewis blood group antigens defined by monoclonal anti-colon carcinoma antibodies

TL;DR: Five monoclonal antibodies directed against human cancer cells were prepared by the murine hybridoma technique and one of the anti-Leb antibodies cross-reacts with blood group H glycolipid and has binding properties similar to those of the previously described antibody NS-10-17.
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Characterization and Functional Analysis of the Human Inducible Nitric Oxide Synthase Gene Promoter

TL;DR: Despite the significant homology between the human and mouse inducible nitric oxide synthase promoter sequence, control of the two genes is quite different and the murine promoter responds increasingly to cytokines that are not effective for the human analog.
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Phosphorylation of Rabies Virus Nucleoprotein Regulates Viral RNA Transcription and Replication by Modulating Leader RNA Encapsidation

TL;DR: It is found that unphosphorylated N binds more strongly to leader RNA than the phosphorylation N; however, the rates of transcription and replication of the rabies virus minigenome were significantly lower with the unph phosphate-based N than with the phosphorieslated N.
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Subacute sclerosing panencephalitis: isolation of a virus encephalitogenic for ferrets.

TL;DR: This encephalitis, characterized by abnormal electroencephalographic (EEG) patterns and histologic lesions, was successfully transmitted serially in ferrets by intracerebral subinoculation of suspensions of ferret brain tissue.