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Hiroshi Yamamoto

Researcher at National Institutes of Natural Sciences, Japan

Publications -  1127
Citations -  17978

Hiroshi Yamamoto is an academic researcher from National Institutes of Natural Sciences, Japan. The author has contributed to research in topics: Medicine & Thin film. The author has an hindex of 62, co-authored 1067 publications receiving 16624 citations. Previous affiliations of Hiroshi Yamamoto include Saitama University & Tokyo Institute of Technology.

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Streptozotocin and alloxan induce DNA strand breaks and poly(ADP-ribose) synthetase in pancreatic islets.

TL;DR: It is described that both Streptozotocin and alloxan cause DNA strand breaks which stimulate nuclear poly(ADP–ribose) synthetase, thereby depleting intracellular NAD and inhibiting proinsulin synthesis in isolated pancreatic islets of rats.
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Highly mobile gapless excitations in a two-dimensional candidate quantum spin liquid.

TL;DR: The thermal conductivity of a recently identified candidate quantum spin liquid candidate, the organic insulator EtMe3Sb[Pd(dmit)2]2, is measured, indicating the presence of gapless excitations with an extremely long mean free path, analogous to excitations near the Fermi surface in pure metals.
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High preoperative neutrophil-lymphocyte ratio predicts poor survival in patients with gastric cancer

TL;DR: A high preoperative NLR may be a convenient biomarker to identify patients with a poor prognosis after resection for primary gastric cancer, and was an independent risk factor for reduced survival on multivariate analysis.
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Correlation between Long-Term Survival in Breast Cancer Patients and Amplification of Two Putative Oncogene-Coamplification Units: hst-1/int-2 and c-erbB-2/ear-1

TL;DR: Cox's life-table regression analysis showed that amplification of c-erbB-2 had a prognostic value, which was independent of other known prognostic factors such as lymph node status and tumor size.
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Effects of obstructive sleep apnea on circulating ICAM-1, IL-8, and MCP-1

TL;DR: It is observed that nCPAP decreased apnea, desaturation, and the circulating ICAM-1 and IL-8 levels in OSAS patients, and observations suggest that n CPAP therapy could reduce OSAS-induced hypoxia and generation of inflammatory mediators.