H
Hong Li
Researcher at Chongqing University
Publications - 70
Citations - 2257
Hong Li is an academic researcher from Chongqing University. The author has contributed to research in topics: Chemistry & Adsorption. The author has an hindex of 18, co-authored 60 publications receiving 1135 citations. Previous affiliations of Hong Li include Chinese Academy of Sciences.
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Journal ArticleDOI
Phytoplankton response to polystyrene microplastics: Perspective from an entire growth period.
Yufeng Mao,Hainan Ai,Yi Chen,Zhenyu Zhang,Peng Zeng,Li Kang,Wei Li,Weikang Gu,Qiang He,Hong Li +9 more
TL;DR: It is confirmed that polystyrene microplastics can impair but then enhance algae growth, which will be helpful in understanding the ecological risks of microplastic.
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Calcium-rich biochar from the pyrolysis of crab shell for phosphorus removal.
TL;DR: Calcium-rich biochars prepared through pyrolysis of crab shell at various temperatures were characterized for physicochemical properties and P removal potential and results suggested that CRB was competent for P removal/recovery from wastewater.
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Reducing the recruitment of sedimented algae and nutrient release into the overlying water using modified soil/sand flocculation-capping in eutrophic lakes.
TL;DR: The study suggested that by using MLS-capping technology it is possible to quickly reduce the nutrient and turbidity of water by flocculating and capping the algal cells into the sediment, where the resuspension of algal flocs is physically reduced and the diffusion of nutrients from sediment to the overlying water chemically blocked by the MLS capping layers.
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Nanoplastics display strong stability in aqueous environments: Insights from aggregation behaviour and theoretical calculations.
TL;DR: The results highlight the high stability of nano-PS in aquatic environments, which could help facilitate the evaluation of their environmental impact.
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Cytotoxic effects of polystyrene nanoplastics with different surface functionalization on human HepG2 cells.
TL;DR: The cytotoxic effects of PS-NPs on HepG2 cells were defined and it was demonstrated that the cellular oxidative damage occurred and that the antioxidation enzymes may not be able to maintain the balance between the generation of oxidant species and the antioxidant defense.