H
Huamei Chen
Researcher at South China Agricultural University
Publications - 3
Citations - 42
Huamei Chen is an academic researcher from South China Agricultural University. The author has contributed to research in topics: Gene & Downregulation and upregulation. The author has an hindex of 1, co-authored 3 publications receiving 6 citations.
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Comparative physiological and transcriptomic analyses illuminate common mechanisms by which silicon alleviates cadmium and arsenic toxicity in rice seedlings.
Huiqiong Chen,Xiaoyu Liang,Xiaomei Gong,John R. Reinfelder,Huamei Chen,Chongjun Sun,Xiulian Liu,Shuchang Zhang,Fangbai Li,Chuanping Liu,Junliang Zhao,Jicai Yi +11 more
TL;DR: The transcriptomes of the Cd- and As-stressed rice roots were similarly and profoundly reshaped by Si application, suggesting that Si may play a fundamental, active role in plant defense against heavy metal/loid stresses by modulating whole genome expression.
Journal ArticleDOI
Comparative transcriptomics provide new insights into the mechanisms by which foliar silicon alleviates the effects of cadmium exposure in rice
Junliang Zhao,Chongjun Sun,Xiaoyu Liang,Xiaomei Gong,Huamei Chen,Xiulian Liu,Shuchang Zhang,Fangbai Li,Junliang Zhao,Jicai Yi +9 more
TL;DR: In this paper, the beneficial effects of foliar silicon in an indica rice Huanghuazhan (HHZ) were investigated, and it was shown that foliar Si increases the dry weight and decreases Cd translocation in Cd-exposed rice at the grain-filling stage only, implying that the filling stage is critical for foliar si to reduce Cd accumulation.
Journal ArticleDOI
Genetic Mapping and Transcriptomic Analysis Revealed the Molecular Mechanism Underlying Leaf-Rolling and a Candidate Protein Phosphatase Gene for the Rolled Leaf-Dominant (RL-D) Mutant in Rice
TL;DR: This study presents a novel dominant rolled-leaf germplasm that may help to improve rice leaf morphology in the future and suggests that the RL-D phenotype might result from abnormal sclerenchyma tissue development, possibly regulated by OsPP2C via the dephosphorylation pathway.