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Ilan S. Wittstein

Researcher at Johns Hopkins University

Publications -  64
Citations -  7882

Ilan S. Wittstein is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Heart failure & Cardiomyopathy. The author has an hindex of 27, co-authored 58 publications receiving 6737 citations. Previous affiliations of Ilan S. Wittstein include Johns Hopkins University School of Medicine.

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Neurohumoral Features of Myocardial Stunning Due to Sudden Emotional Stress

TL;DR: Emotional stress can precipitate severe, reversible left ventricular dysfunction in patients without coronary disease andaggerated sympathetic stimulation is probably central to the cause of this syndrome.
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International Expert Consensus Document on Takotsubo Syndrome (Part I): Clinical Characteristics, Diagnostic Criteria, and Pathophysiology

TL;DR: The clinical expert consensus document part I summarizes the current state of knowledge on clinical presentation and characteristics of TTS and agrees on controversies surrounding TTS such as nomenclature, different TTS types, role of coronary artery disease, and etiology.
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The cardiovascular toll of stress

TL;DR: An overview of the association between stress and cardiovascular morbidity is provided, the mechanisms for this association are discussed, and possible therapeutic implications are addressed.
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International Expert Consensus Document on Takotsubo Syndrome (Part II): Diagnostic Workup, Outcome, and Management

TL;DR: The clinical expert consensus statement on takotsubo syndrome (TTS) part II focuses on the diagnostic workup, outcome, and management and summarizes the diagnostic approach, which may facilitate correct and timely diagnosis.
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Stress Cardiomyopathy After Intravenous Administration of Catecholamines and Beta-Receptor Agonists

TL;DR: Exposure to catecholamines and beta-receptor agonists used routinely during procedures and diagnostic tests can precipitate all the features of stress cardiomyopathy, including cardiac isoenzyme elevation, QTc interval prolongation, and rapidly reversible cardiac dysfunction.