T
Trinity J. Bivalacqua
Researcher at Johns Hopkins University
Publications - 543
Citations - 18459
Trinity J. Bivalacqua is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Bladder cancer & Cystectomy. The author has an hindex of 56, co-authored 498 publications receiving 15806 citations. Previous affiliations of Trinity J. Bivalacqua include National Institutes of Health & University Medical Center New Orleans.
Papers
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Journal ArticleDOI
Neurohumoral Features of Myocardial Stunning Due to Sudden Emotional Stress
Ilan S. Wittstein,David R. Thiemann,Joao A.C. Lima,Kenneth L. Baughman,Steven P. Schulman,Gary Gerstenblith,Katherine C. Wu,Jeffrey J. Rade,Trinity J. Bivalacqua,Hunter C. Champion +9 more
TL;DR: Emotional stress can precipitate severe, reversible left ventricular dysfunction in patients without coronary disease andaggerated sympathetic stimulation is probably central to the cause of this syndrome.
Journal ArticleDOI
RhoA/Rho-kinase suppresses endothelial nitric oxide synthase in the penis: A mechanism for diabetes-associated erectile dysfunction
Trinity J. Bivalacqua,Hunter C. Champion,Mustafa F. Usta,Selim Cellek,Kanchan Chitaley,R. Clinton Webb,Ronald L. Lewis,Thomas M. Mills,Wayne J.G. Hellstrom,Philip J. Kadowitz +9 more
TL;DR: Data imply that inhibition of RhoA/Rho-kinase improves eNOS protein content and activity thus restoring erectile function in diabetes, demonstrating a previously undescribed mechanism for the down-regulation of penile eNos in diabetes.
Journal ArticleDOI
Priapism: Pathogenesis, Epidemiology, and Management
Gregory A. Broderick,Ates Kadioglu,Trinity J. Bivalacqua,Hussein Ghanem,Ajay Nehra,Rany Shamloul,Rany Shamloul,Rany Shamloul +7 more
TL;DR: Broderick et al. as mentioned in this paper provided a comprehensive review of the evidence-based guidelines written over the past decade and reviewed the priapism literature from 2003 to 2009, concluding that prompt diagnosis and appropriate management are necessary to spare patients ineffective interventions and maximize erectile function outcomes.
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Cavernous neurotomy causes hypoxia and fibrosis in rat corpus cavernosum.
Somboon Leungwattanakij,Trinity J. Bivalacqua,Mustafa F. Usta,Dae Yul Yang,Jae Seog Hyun,Hunter C. Champion,Asim B. Abdel-Mageed,Wayne J.G. Hellstrom +7 more
TL;DR: An increase in TGF-beta(1), HIF-1alpha, and collagen III synthesis in rat cavernosal smooth musculature after cavernous neurotomies is demonstrated, demonstrating reduced cavernous fibrosis by employing various vasoactive agents or interventions that increase oxygenation to the corporal tissues during the postoperative period.
Journal ArticleDOI
Hemolysis in sickle cell mice causes pulmonary hypertension due to global impairment in nitric oxide bioavailability
Lewis L. Hsu,Lewis L. Hsu,Hunter C. Champion,Sally Campbell-Lee,Trinity J. Bivalacqua,Elizabeth A. Manci,Bhalchandra A. Diwan,Daniel Schimel,Audrey E. Cochard,Xunde Wang,Alan N. Schechter,Constance Tom Noguchi,Mark T. Gladwin +12 more
TL;DR: It is demonstrated that hemolytic anemia is sufficient to produce endothelial dysfunction and global dysregulation of NO and to cause vasculopathy in sickle mice.