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Ingrid Stroo

Researcher at University of Amsterdam

Publications -  29
Citations -  1179

Ingrid Stroo is an academic researcher from University of Amsterdam. The author has contributed to research in topics: Inflammation & Kidney. The author has an hindex of 16, co-authored 29 publications receiving 1032 citations.

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Correction: Corrigendum: Effect of TREM-1 blockade and single nucleotide variants in experimental renal injury and kidney transplantation

TL;DR: This research presents a novel probabilistic procedure that allows us to assess the importance of knowing the carrier and removal status of canine coronavirus in the context of infectious disease.
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Depletion of Gut Microbiota Protects against Renal Ischemia-Reperfusion Injury

TL;DR: It is shown that depletion of gut microbiota profoundly protects against renal ischemia-reperfusion (I/R) injury by reducing maturation status of F4/80+ renal resident macrophages and BM monocytes and dampening the inflammatory response by targeting microbiota-derived mediators might be a promising therapy against I/R injury.
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Chemokine expression in renal ischemia/reperfusion injury is most profound during the reparative phase

TL;DR: In this paper, the authors performed microarray analyses to identify chemokines that play a role during the inflammatory and repair phase after renal ischemia/reperfusion (I/R) injury and investigated the temporal relationship between chemokine expression, leukocyte accumulation and renal damage/repair.
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Lipopolysaccharide inhibits Th2 lung inflammation induced by house dust mite allergens in mice

TL;DR: This study is the first to provide insights into the effects of LPS in an allergic lung inflammation model making use of a clinically relevant allergen without a systemic adjuvant, revealing that LPS dose-dependently inhibits HDM-induced pulmonary Th2 responses.
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SDF-1 provides morphological and functional protection against renal ischaemia/reperfusion injury

TL;DR: In this paper, the role of SDF-1 in renal ischaemia/reperfusion injury by locally decreasing SDF1 expression and subsequent signalling in the corticomedullary region of the kidney using antisense oligonucleotide treatment in mice.