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Inna Tabansky
Researcher at Rockefeller University
Publications - 37
Citations - 651
Inna Tabansky is an academic researcher from Rockefeller University. The author has contributed to research in topics: Interstitial cystitis & Medicine. The author has an hindex of 10, co-authored 31 publications receiving 532 citations. Previous affiliations of Inna Tabansky include New York University & Howard Hughes Medical Institute.
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Multi-trait evolution in a cave fish, Astyanax mexicanus.
Meredith E. Protas,Inna Tabansky,Melissa D. Conrad,Joshua B. Gross,Oriol Vidal,Clifford J. Tabin,Richard Borowsky +6 more
TL;DR: In this paper, quantitative trait loci (QTL) mapping of 12 traits that differ significantly between the recently evolved Mexican cave tetra and its surface conspecific was investigated through QTL mapping.
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Developmental Bias in Cleavage-Stage Mouse Blastomeres
Inna Tabansky,Alan B. Lenarcic,Karine Loulier,Derin B. Keskin,Jacqueline Rosains,José Rivera-Feliciano,José Rivera-Feliciano,Jeff W. Lichtman,Jean Livet,Joel N. H. Stern,Joshua R. Sanes,Kevin Eggan +11 more
TL;DR: The Rainbow transgenic mice described here have allowed us to detect lineage-dependent bias in early development and enable assessment of the developmental equivalence of mammalian progenitor cells in a variety of tissues.
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Single-Cell Analysis Uncovers Clonal Acinar Cell Heterogeneity in the Adult Pancreas
Damian Wollny,Sheng Zhao,Isabelle Everlien,Xiao-Kang Lun,Jan Brunken,Daniel Brüne,Frederik Ziebell,Inna Tabansky,Wilko Weichert,Anna Marciniak-Czochra,Ana Martin-Villalba +10 more
TL;DR: This study identifies a functionally and molecularly distinct acinar subpopulation and thus transforms the understanding of the acinar cell compartment as a pool of equipotent secretory cells.
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Epigenetic changes in the developing brain: Effects on behavior
TL;DR: The purpose of this Sackler Colloquium was to bring together experts in the two fields—epigenetic chemistry and behavioral neuroscience—in the spirit of mutual education.
Journal ArticleDOI
Targeting DEC-205-DCIR2+ dendritic cells promotes immunological tolerance in proteolipid protein-induced experimental autoimmune encephalomyelitis.
Inna Tabansky,Derin B. Keskin,Derin B. Keskin,Deepika Watts,Cathleen Petzold,Michael Funaro,Michael Funaro,Warren Sands,Paul Wright,Edmond J. Yunis,Souhel Najjar,Betty Diamond,Yonghao Cao,Yonghao Cao,David J. Mooney,Karsten Kretschmer,Joel N. H. Stern,Joel N. H. Stern,Joel N. H. Stern +18 more
TL;DR: These results suggest that multiple mechanisms can lead to the expansion of the Treg population, depending on the DC subset and receptor targeted, and provide evidence that the induction of antigen-specific T cell tolerance is not a unique property of CD11c+CD8+DEC-205+ DCs.