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Ishnoor Kaur

Researcher at Chitkara University

Publications -  30
Citations -  444

Ishnoor Kaur is an academic researcher from Chitkara University. The author has contributed to research in topics: Medicine & Disease. The author has an hindex of 7, co-authored 22 publications receiving 146 citations.

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The dual impact of ACE2 in COVID-19 and ironical actions in geriatrics and pediatrics with possible therapeutic solutions.

TL;DR: A review of possible therapeutic interventions, targeting both the protective and deleterious effects of ACE2 in COVID-19 disease, primarily inhibiting ACE2-virus interactions or administering soluble ACE2 is demonstrated.
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The Interplay of ABC Transporters in Aβ Translocation and Cholesterol Metabolism: Implicating Their Roles in Alzheimer's Disease.

TL;DR: The authors aim to establish the significance of this alternative approach as a novel therapeutic target in AD, to provide the researchers an opportunity to evaluate the potential aspects of ABC transporters in AD treatment.
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The dichotomy of nanotechnology as the cutting edge of agriculture: Nano-farming as an asset versus nanotoxicity

TL;DR: In this article, the authors aim to provide a significant opportunity to the nanotech researchers, botanists and environmentalists to promote judicial use of nanoparticles and establish a secure and safe environment.
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Exploring the Potential of Therapeutic Agents Targeted towards Mitigating the Events Associated with Amyloid-β Cascade in Alzheimer's Disease.

TL;DR: The authors aim to highlight the treatment perspective, targeting the amyloid hypothesis, while simultaneously emphasizing the need to conduct further investigations, in order to provide an opportunity to neurologists to develop novel and reliable treatment therapies for the retardation of AD progression.
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Role of metallic pollutants in neurodegeneration: effects of aluminum, lead, mercury, and arsenic in mediating brain impairment events and autism spectrum disorder

TL;DR: In this article, a review focused on the primary metallic components present in the environment (aluminum, lead, mercury, and arsenic) responsible for accelerating ASD symptoms by a set of general mechanisms like oxidative stress reduction, glycolysis suppression, microglial activation, and metalloprotein disruption, resulting in apoptotic signaling, neurotoxic effects, and neuroinflammatory responses.