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Ivo Šteiner

Researcher at Charles University in Prague

Publications -  41
Citations -  2872

Ivo Šteiner is an academic researcher from Charles University in Prague. The author has contributed to research in topics: Aortic valve & Bicuspid valve. The author has an hindex of 11, co-authored 38 publications receiving 2501 citations.

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Human papillomavirus genotype attribution in invasive cervical cancer: a retrospective cross-sectional worldwide study

Silvia de Sanjosé, +62 more
- 01 Nov 2010 - 
TL;DR: HPV types 16, 18, 31, 33, 35, 45, 52, and 58 should be given priority when the cross-protective effects of current vaccines are assessed, and for formulation of recommendations for the use of second-generation polyvalent HPV vaccines, according to this largest assessment of HPV genotypes to date.
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Prognostic significance of CD3+ tumor-infiltrating lymphocytes in ovarian carcinoma.

TL;DR: Investigation of the prognostic significance of CD3+ tumor-infiltrating T lymphocytes (TIL) on overall survival of EOC patients indicates that the intraepithelial CD3- TIL count is a significant prognostic factor in EOC.
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Lymphatic vasculature is increased in heart valves, ischaemic and inflamed hearts and in cholesterol-rich and calcified atherosclerotic lesions

TL;DR: In this paper, the highest number of lymphatics were found in valves in infective endocarditis, where they accounted nearly 100% of all vessels in certain areas, whereas inflammatory cell-rich areas were more prone to angiogenesis.
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Bone formation in cardiac valves: a histopathological study of 128 cases.

TL;DR: In a series of 1,177 patients with surgical valve resection, there were 128 patients with the histological finding of bone and/or cartilage formation (bony metaplasia; heterotopic ossification) in the excised valve.
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Calcific aortic valve stenosis: Immunohistochemical analysis of inflammatory infiltrate

TL;DR: The findings revealed that in CAS, there were chronic inflammatory features with infiltrates comprising lymphocytes, polyclonal plasma cells, histiocytes and mast cells, and CAS did not fulfill the criteria of the recently described clinicopathological entity IgG4-related sclerosing systemic disease.