J
J.L. Molinari
Researcher at National Autonomous University of Mexico
Publications - 7
Citations - 282
J.L. Molinari is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Taenia solium & Concanavalin A. The author has an hindex of 6, co-authored 7 publications receiving 276 citations.
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Immunization against porcine cysticercosis in an endemic area in Mexico: a field and laboratory study.
TL;DR: An antigenic extract from Taenia solium metacestodes was evaluated for immunogenicity in pig populations from a large area of endemic porcine cysticercosis in the State of Guerrero, Mexico and Histopathologic studies revealed granuloma formation surrounding the metacstodes of the immunized Cysticercotic and cysticERCotic immunized pigs.
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Field trial for reducing porcine Taenia solium cysticercosis in Mexico by systematic vaccination of pigs.
TL;DR: A statistically significant decline in the prevalence of porcine cysticercosis was observed at the end of the trial, and it may be possible to turn a susceptible pig population into a protected one by systematic vaccination.
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Taenia solium: immunity in hogs to the Cysticercus.
TL;DR: Histopathological studies showed that more than 40% of larvae obtained from immunized hogs were completely destroyed and the others were seen in various stages of degeneration.
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Suppression of murine lymphocyte proliferation induced by a small RNA purified from the Taenia solium metacestode
TL;DR: The results suggest that this substance is a RNA-peptide molecule whose RNA moiety accounts for its suppressive activity and the findings suggest that in vivo the factor may be a modulator of the immune response.
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Immune response impairment, genotoxicity and morphological transformation induced by Taenia solium metacestode.
Luis A. Herrera,Pilar Santiago,Gloria Rojas,Paz Maria Salazar,Patricia Tato,J.L. Molinari,Dietmar Schiffmann,Patricia Ostrosky-Wegman +7 more
TL;DR: The results strongly suggest that the parasite produces genetic instability in the host cells, which could result in immunosuppression and malignant transformation of target cells.