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J. Steven Reznick

Researcher at University of North Carolina at Chapel Hill

Publications -  54
Citations -  5260

J. Steven Reznick is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: Autism & Cognition. The author has an hindex of 30, co-authored 52 publications receiving 4745 citations.

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Visual Scanning of Faces in Autism

TL;DR: The visual scanpaths of five high-functioning adult autistic males and five adult male controls were recorded using an infrared corneal reflection technique as they viewed photographs of human faces to suggest a mechanism that may subserve the social information processing deficits that characterize autism spectrum disorders.
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Short-form versions of the MacArthur Communicative Development Inventories.

TL;DR: The MacArthur Communicative Development Inventories (CDI) as mentioned in this paper are a pair of widely used parent-report instruments for assessing communicative skills in infants and toddlers, and they have been widely used in clinical applications.
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The broad autism phenotype questionnaire.

TL;DR: A new instrument designed to measure the broad autism phenotype in adults, the Broad Autism Phenotype Questionnaire (BAPQ), was administered to 86 parents of autistic individuals and 64 community control parents and found sensitivity and specificity of the BAPQ for detecting the B AP were high.
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The effects of socioeconomic status, race, and parenting on language development in early childhood.

TL;DR: The results highlight the importance of sensitive parenting and suggest that the association between negative-intrusive parenting and language development may depend upon family context.
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White Matter Microstructure and Atypical Visual Orienting in 7-Month-Olds at Risk for Autism

TL;DR: Visual orienting latencies were longer in 7-month-old infants who expressed ASD symptoms at 25 months compared with both high-risk negative infants and low-risk infants, and abnormal functional specialization of posterior cortical circuits directly informs a novel model of ASD pathogenesis.