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Jaap W. Deckers

Researcher at Erasmus University Rotterdam

Publications -  273
Citations -  39450

Jaap W. Deckers is an academic researcher from Erasmus University Rotterdam. The author has contributed to research in topics: Myocardial infarction & Heart failure. The author has an hindex of 68, co-authored 268 publications receiving 37766 citations. Previous affiliations of Jaap W. Deckers include European Society of Cardiology & Hannover Medical School.

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Consistency of heart rate–QTc prolongation consistency and sudden cardiac death: The Rotterdam Study

TL;DR: The results suggest that repeated measurements of the QTc interval could enhance risk stratification, and persons with a consistently prolonged QTC interval did have a higher risk of SCD.
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Efficacy of perindopril in reducing risk of cardiac events in patients with revascularized coronary artery disease

TL;DR: In this article, the effect of adding perindopril 8 mg once daily to standard preventive therapy in the subgroup of EUROPA patients with previous revascularization and without previous myocardial infarction (MI) was assessed.
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Incidence of cardiovascular events in patients with stabilized coronary heart disease: the EUROASPIRE IV follow-up study.

TL;DR: In patients with stabilized CHD, comorbid conditions that may reflect the ubiquitous nature of atherosclerosis, dominate lifestyle-related and other modifiable risk factors in terms of prognosis, at least over a 2-year follow-up period.
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Progression and regression of the atherosclerotic plaque

TL;DR: Angiographic trials demonstrated that retardation or arrest of coronary atherosclerosis was possible after an intervention, but the ultimate goal, regression of the lesion, was only achieved in a small number of patients, which is a considerable achievement for those patients with coronary Atherosclerosis.
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ACE inhibition with perindopril and biomarkers of atherosclerosis and thrombosis: Results from the PERTINENT study

TL;DR: The PERTINENT trial observed significant effects of ACE inhibition on biomarkers of the atherothrombotic complications (D-dimer) and the proinflammatory cytokine TNF-alpha, but not on biomarker of inflammation associated with atherosclerosis (CRP and fibrinogen).