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James S. Ballantyne

Researcher at University of Guelph

Publications -  112
Citations -  3888

James S. Ballantyne is an academic researcher from University of Guelph. The author has contributed to research in topics: Urea & Na+/K+-ATPase. The author has an hindex of 34, co-authored 112 publications receiving 3666 citations. Previous affiliations of James S. Ballantyne include University of St Andrews & University of Ottawa.

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Reciprocal expression of gill Na+/K+-ATPase alpha-subunit isoforms alpha1a and alpha1b during seawater acclimation of three salmonid fishes that vary in their salinity tolerance.

TL;DR: The reciprocal expression of Na+/K+-ATPase isoforms α1a and α1b during seawater acclimation suggests they may have different roles in the gills of freshwater and marine fishes; ion uptake in freshwater fish and ion secretion in marine fishes.
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Jaws: The Inside Story. The Metabolism of Elasmobranch Fishes

TL;DR: The reduced availability of glutamine coupled with the low levels of nonesterified fatty acids in the blood reduces the availability of molecules essential for tumor growth, which may provide marine elasmobranchs with a “systemic” resistance to cancer.
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High stocking density alters the energy metabolism of brook charr, Salvelinus fontinalis

TL;DR: It is suggested that high stocking density has the effect of mobilizing triglyceride sources, promoting gluconeogenesis from glycerol, but has little effect on protein metabolism.
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Cortisol-induced changes in some aspects of the intermediary metabolism of Salvelinus fontinalis

TL;DR: Cortisol was administered to brook charr (Salvelinus fontinalis) in the form of slow-release intraperitoneal implants to investigate the effect of chronic cortisol stimulation (up to 60 days) on various aspects of intermediary metabolism, and to determine whether such cortisol-induced changes were comparable to those seen in chronically fasted charr as mentioned in this paper.
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Active urea transport and an unusual basolateral membrane composition in the gills of a marine elasmobranch

TL;DR: This work proposes that a unique combination of active urea transport and modification of the phospholipid bilayer membrane is responsible for decreasing the gill permeability to urea and facilitating urea retention by the gills of Squalus acanthias.