J
Jennifer J. Bell
Researcher at Columbia University
Publications - 23
Citations - 2045
Jennifer J. Bell is an academic researcher from Columbia University. The author has contributed to research in topics: Osteoporosis & Bone mineral. The author has an hindex of 16, co-authored 23 publications receiving 1924 citations. Previous affiliations of Jennifer J. Bell include University of California, San Francisco.
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Increased Bone Mass as a Result of Estrogen Therapy in a Man with Aromatase Deficiency
TL;DR: The sex steroids are critically important in helping to establish peak bone mass for both sexes and the more bone mass one gains in the formative years, the less likely it is that increased bone resorption and decreased bone formation will result in osteoporosis.
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Neonatal hyperbilirubinemia and Rhesus disease of the newborn: incidence and impairment estimates for 2010 at regional and global levels
Vinod K. Bhutani,Alvin Zipursky,Hannah Blencowe,Rajesh Khanna,Michael Sgro,Finn Ebbesen,Jennifer J. Bell,Rintaro Mori,Tina M. Slusher,Nahed Fahmy,Vinod K. Paul,Lizhong Du,A. A. Okolo,Maria Fernanda Branco de Almeida,Bolajoko O. Olusanya,Praveen Kumar,Simon Cousens,Joy E Lawn +17 more
TL;DR: Failure to prevent Rh sensitization and manage neonatal hyperbilirubinemia results in 114,100 avoidable neonatal deaths and many children grow up with disabilities.
Journal ArticleDOI
Gender change from female to male in classical congenital adrenal hyperplasia.
Heino F. L. Meyer-Bahlburg,Heino F. L. Meyer-Bahlburg,Rhoda Gruen,Rhoda Gruen,Maria I. New,Jennifer J. Bell,Akira Morishima,Mona Shimshi,Yvette Bueno,Ileana Vargas,Susan W. Baker +10 more
TL;DR: The most plausible factors contributing to cross-gender identity development in these patients appeared to be neither a particular genotype or endocrinotype nor a sex-typing bias on the part of the parents but a combination of a gender-atypical behavioral self-image, aGender- atypical body image, and the development of erotic attraction to women.
Journal ArticleDOI
Increased bone mass as a result of estrogen therapy in a man with aromatase deficiency
TL;DR: The failure to achieve optimal peak bone mass is a pathogenetic mechanism in osteoporosis as discussed by the authors, since the aging process is associated with bone loss, the more bone mass one gains in the formative years, the less likely it is that increased bone resorption and decreased bone formation will result in osteopsorosis.
Journal ArticleDOI
Clinical studies with recombinant-dna-derived methionyl human growth hormone in growth hormone deficient children
S. L. Kaplan,G. P. August,S. L. Blethen,D. R. Brown,Raymond L. Hintz,A. Johansen,Leslie P. Plotnick,Louis E. Underwood,Jennifer J. Bell,Robert M. Blizzard,Thomas P. Foley,N. J. Hopwood,R. T. Kirkland,Ron G. Rosenfeld,J J Van Wyk +14 more
TL;DR: Thirty-six children with growth hormone deficiency were treated for up to 48 months with methionyl human growth hormone (hGH) synthesised by DNA recombinant methods and the growth rate increased, similar to the effect of pituitary hGH in ten GH deficient children.