scispace - formally typeset
J

Jennifer L. Jenner

Researcher at Tufts University

Publications -  29
Citations -  4719

Jennifer L. Jenner is an academic researcher from Tufts University. The author has contributed to research in topics: Cholesterol & Lipoprotein(a). The author has an hindex of 25, co-authored 28 publications receiving 4609 citations. Previous affiliations of Jennifer L. Jenner include United States Department of Agriculture & University of Washington.

Papers
More filters
Journal ArticleDOI

Familial lipoprotein disorders in patients with premature coronary artery disease.

TL;DR: It is indicated that more than half of patients with premature CAD have a familial lipoprotein disorder, with Lp(a) excess, hypertriglyceridemia with hypoalphalipoproteinemia, and combined hyperlipidemia and apolipoprotein levels are in part genetically determined.
Journal ArticleDOI

Low density lipoprotein particle size and coronary artery disease.

TL;DR: The difference in LDL particle size between CAD patients and controls was not independent but was highly associated with elevated triglyceride levels and decreased high density lipoprotein (HDL) cholesterol levels.
Journal ArticleDOI

Lipoprotein(a) levels and risk of coronary heart disease in men. The lipid Research Clinics Coronary Primary Prevention Trial

TL;DR: The data are consistent with the concept that an elevated Lp(a) level is an independent risk factor for CHD in hypercholesterolemic white men.
Journal ArticleDOI

Elevated plasma lipoprotein(a) and coronary heart disease in men aged 55 years and younger : A prospective study

TL;DR: Elevated plasma Lp(a) is an independent risk factor for the development of premature CHD in men, comparable in magnitude and prevalence (ie, attributable risk) to a total cholesterol level of 6.2 mmol/L (240 mg/dL) or more, or an HDL level less than 0.3%.
Journal ArticleDOI

Lipoprotein cholesterol, apolipoprotein A-I and B and lipoprotein (a) abnormalities in men with premature coronary artery disease.

TL;DR: Stepwise discriminant analysis indicates that smoking, hypertension, decreased apolipoprotein A-I, increased apoliprotein B, increased Lp(a) and diabetes are all significant factors in descending order of importance in distinguishing patients with coronary artery disease from normal control subjects.