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Jia Qi

Researcher at Shanghai Jiao Tong University

Publications -  39
Citations -  2057

Jia Qi is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Glutamatergic & Ventral tegmental area. The author has an hindex of 17, co-authored 33 publications receiving 1691 citations. Previous affiliations of Jia Qi include Shenyang Pharmaceutical University & National Institute on Drug Abuse.

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Dopaminergic and glutamatergic microdomains in a subset of rodent mesoaccumbens axons

TL;DR: A complex type of signaling by mesoaccumbens fibers is revealed in which dopamine and glutamate can be released from the same axons, but are not normally released at the same site or from theSame synaptic vesicles.
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Role of Glutamatergic Projections from Ventral Tegmental Area to Lateral Habenula in Aversive Conditioning

TL;DR: There is a glutamatergic signal from VTA VGluT2-mesohabenular neurons that plays a role in aversion by activating LHb glutamatorgic receptors.
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VTA glutamatergic inputs to nucleus accumbens drive aversion by acting on GABAergic interneurons

TL;DR: The ventral tegmental area (VTA) is best known for its dopamine neurons, some of which project to nucleus accumbens (nAcc), but the VTA also has glutamatergic neurons that project to nAcc, and the mesoaccumbens glutamatorgic pathways are reported on, which are the first glutamaters to be shown to mediate aversion instead of reward.
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A glutamatergic reward input from the dorsal raphe to ventral tegmental area dopamine neurons.

TL;DR: Findings indicate that the DR-VGluT3 pathway to VTA utilizes glutamate as a neurotransmitter and is a substrate linking the DR—one of the most sensitive reward sites in the brain—to VTA dopaminergic neurons.
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Painful pathways induced by TLR stimulation of dorsal root ganglion neurons.

TL;DR: Stimulation of peripheral neurons by TLR ligands can induce nerve pain, and in vivo administration of a TLR9 antagonist, known as a suppressive oligodeoxynucleotide, blocked tumor-induced temperature sensitivity.