J
Jin Tae Hong
Researcher at Chungbuk National University
Publications - 576
Citations - 17141
Jin Tae Hong is an academic researcher from Chungbuk National University. The author has contributed to research in topics: Cancer cell & Apoptosis. The author has an hindex of 56, co-authored 543 publications receiving 14046 citations. Previous affiliations of Jin Tae Hong include Korea Research Institute of Bioscience and Biotechnology.
Papers
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Neuro-inflammation induced by lipopolysaccharide causes cognitive impairment through enhancement of beta-amyloid generation
Jae Woong Lee,Yong Kyung Lee,Dong Yeon Yuk,Dong Young Choi,Sang Bae Ban,Ki Wan Oh,Jin Tae Hong +6 more
TL;DR: This study suggests that neuro-inflammatory reaction could contribute to AD pathology, and anti-inflammatory agent could be useful for the prevention of AD.
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Therapeutic application of anti-arthritis, pain-releasing, and anti-cancer effects of bee venom and its constituent compounds.
TL;DR: Bee venom (BV) therapy (BVT), the therapeutic application of BV, has been used in traditional medicine to treat diseases, such as arthritis, rheumatism, pain, cancerous tumors, and skin diseases, and nonpeptide components which have a variety of pharmaceutical properties.
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Roles of NF-κB in Cancer and Inflammatory Diseases and Their Therapeutic Approaches.
Mi Hee Park,Jin Tae Hong +1 more
TL;DR: In this article, the authors proposed to inhibit NF-κB signaling in cancer and inflammatory diseases, such as rheumatoid arthritis, atherosclerosis, inflammatory bowel diseases, multiple sclerosis and malignant tumors.
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Therapeutic applications of compounds in the Magnolia family
Young-Jung Lee,Yoot Mo Lee,Yoot Mo Lee,Chong-Kil Lee,Jae-Kyung Jung,Sang-Bae Han,Jin Tae Hong +6 more
TL;DR: The aim of this study was to review a variety of experimental and clinical reports and, describe the effectiveness, toxicities and pharmacokinetics, and possible mechanisms of Magnolia and/or its constituents.
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Inflammation and Alzheimer's disease.
TL;DR: This review is to assess the association between neuroinflammation and AD through discussion of a variety of experimental and clinical studies on AD and to review treatment strategies designed to treat or prevent AD.