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Jinbo Yang

Researcher at Lanzhou University

Publications -  61
Citations -  2975

Jinbo Yang is an academic researcher from Lanzhou University. The author has contributed to research in topics: Medicine & STAT3. The author has an hindex of 19, co-authored 45 publications receiving 2633 citations. Previous affiliations of Jinbo Yang include Cleveland Clinic Lerner Research Institute & Cleveland Clinic.

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Unphosphorylated STAT3 accumulates in response to IL-6 and activates transcription by binding to NFκB

TL;DR: U-STAT3 sustains cytokine-dependent signaling at late times through a mechanism completely distinct from that used by P-STAT2, and is likely to be important in physiological responses to gp130-linked cytokines and growth factors that activate STAT3, and in cancers that have constitutively active P- STAT3.
Journal Article

Novel roles of unphosphorylated STAT3 in oncogenesis and transcriptional regulation.

TL;DR: Unphosphorylated STAT3, which activates gene expression by a novel mechanism distinct from that used by STAT3 dimers, is very likely to be an important transcription factor both in cancer and in responses to cytokines.
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Roles of unphosphorylated STATs in signaling.

TL;DR: The roles of U-STATs in transcription and regulation of gene expression are discussed, which are distinct from those used by phosphorylated STAT (P-STAT) dimers.
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Reversible methylation of promoter-bound STAT3 by histone-modifying enzymes

TL;DR: It is concluded that the lysine methylation of promoter-bound STAT3 leads to biologically important down-regulation of the dependent responses and that SET9, which is known to help provide an activating methylation mark to H3K4, is recruited to the newly activated SOCS3 promoter by STAT3.
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STAT3 activation in response to IL-6 is prolonged by the binding of IL-6 receptor to EGF receptor

TL;DR: It is shown that prolonged activation of STAT3 results from association of the IL-6 receptor with the epidermal growth factor receptor, which in turn activates STAT3 in a way that is not inhibited by SOCS3, the major negative regulator suppressor of cytokine signaling 3.