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Jiyoti Verma

Researcher at Monash University, Clayton campus

Publications -  8
Citations -  280

Jiyoti Verma is an academic researcher from Monash University, Clayton campus. The author has contributed to research in topics: Candida albicans & RNA polymerase II. The author has an hindex of 6, co-authored 8 publications receiving 196 citations. Previous affiliations of Jiyoti Verma include Indian Institute of Science.

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Glucose Homeostasis Is Important for Immune Cell Viability during Candida Challenge and Host Survival of Systemic Fungal Infection

TL;DR: It is shown that Candida infection perturbs host glucose homeostasis in the murine candidemia model and that glucose supplementation improves host outcomes, supporting the importance of maintaining glucoseHomeostasis for immune cell survival during Candida challenge and for host survival in systemic infection.
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Whole genome expression profiles of yeast RNA polymerase II core subunit, Rpb4, in stress and nonstress conditions

TL;DR: The analysis shows that Rpb4 affects expression of a small yet significant fraction of the genome in both stress and normal conditions, and that on overexpression the stress-specific activators can partially rescue the corresponding defects.
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Wolbachia-mediated virus blocking in mosquito cells is dependent on XRN1-mediated viral RNA degradation and influenced by viral replication rate.

TL;DR: The results suggest that infection with Wolbachia does not inhibit DENV binding or cell entry, but reduces virus replication, and it is observed that WMVB is influenced by virus MOI and rate of virus replication.
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Metabolic competition between host and pathogen dictates inflammasome responses to fungal infection.

TL;DR: In this article, the authors used live cell imaging coupled with a compendium of diverse clinical isolates to define how macrophages respond and activate NLRP3 when faced with the human yeast commensal and pathogen Candida albicans.
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Basal transcription machinery: role in regulation of stress response in eukaryotes.

TL;DR: The logic behind the suggestion that like in prokaryotes, eukaryotes also have a common functional unit in the transcription machinery through which the stress specific transcription factors regulate rapid and highly controlled induction of gene expression associated with generalized stress response is discussed.