J
Jocelyne Bachevalier
Researcher at Yerkes National Primate Research Center
Publications - 146
Citations - 9560
Jocelyne Bachevalier is an academic researcher from Yerkes National Primate Research Center. The author has contributed to research in topics: Hippocampal formation & Hippocampus. The author has an hindex of 51, co-authored 138 publications receiving 8876 citations. Previous affiliations of Jocelyne Bachevalier include University of Texas Health Science Center at San Antonio & Emory University.
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Effects on visual recognition of combined and separate ablations of the entorhinal and perirhinal cortex in rhesus monkeys
TL;DR: The present results demonstrate not only that damage limited to the rhinal cortex is sufficient to produce a severe loss in visual recognition, but also that such damage leads to a far greater loss than damage to any other single structure in the medial part of the temporal lobe.
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Connections of Inferior Temporal Areas TEO and TE with Parietal and Frontal Cortex in Macaque Monkeys
TL;DR: Inferior temporal areas TEO and TE were injected with WGA-HRP and 3H-AA, respectively, or vice versa, in 1-week-old infant and 3-4-year-old adult monkeys (Macaca mulatta), the results indicated that whereas TEO has more extensive connections with parietal areas, TE has more comprehensive connections with prefrontal areas.
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Towards a transgenic model of Huntington’s disease in a non-human primate
Shang Hsun Yang,Pei Hsun Cheng,Heather Banta,Karolina Piotrowska-Nitsche,Karolina Piotrowska-Nitsche,Jin Jing Yang,Eric C.H. Cheng,Brooke R. Snyder,Katherine Larkin,Jun Liu,Jack Orkin,Zhi Hui Fang,Yoland Smith,Jocelyne Bachevalier,Jocelyne Bachevalier,Stuart M. Zola,Shihua Li,Xiao-Jiang Li,Anthony W.S. Chan +18 more
TL;DR: Hallmark features of HD, including nuclear inclusions and neuropil aggregates, were observed in the brains of the HD transgenic monkeys, and the data suggest that it will be feasible to generate valuable non-human primate models of HD and possibly other human genetic diseases.
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The orbitofrontal-amygdala circuit and self-regulation of social-emotional behavior in autism.
TL;DR: Evidence is presented to support the hypothesis that developmental dysfunction of the orbitofrontal-amygdala circuit of the brain is a critical factor in the development of autism and that some of the characteristic deficits of persons with autism in socio-emotional cognition and behavioral self-regulation are related to early dysfunction of different components of this circuit.
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Impaired Safety Signal Learning May be a Biomarker of PTSD
TL;DR: Overall, PTSD subjects demonstrate a lack of safety signal learning and an inability to modulate the fear responses with safety cues, according to a conditional discrimination paradigm used in combat and civilian PTSD populations.