J
John H. Beattie
Researcher at University of Aberdeen
Publications - 117
Citations - 5417
John H. Beattie is an academic researcher from University of Aberdeen. The author has contributed to research in topics: Metallothionein & Zinc. The author has an hindex of 36, co-authored 117 publications receiving 5067 citations. Previous affiliations of John H. Beattie include Rowett Research Institute & Université du Québec à Montréal.
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Journal ArticleDOI
Physiological role of adipose tissue: white adipose tissue as an endocrine and secretory organ
Paul Trayhurn,John H. Beattie +1 more
TL;DR: The key challenges in establishing the secretory functions of white fat are to identify the complement of secreted proteins, to establish the role of each secreted protein, and to assess the pathophysiological consequences of changes in adipocyte protein production with alterations in adiposity.
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Induction, regulation, degradation, and biological significance of mammalian metallothioneins.
TL;DR: The possible biological roles of this protein in mammals are considered, which include participation in cell proliferation and apoptosis, homeostasis of essential metals, cellular free radical scavenging, and metal detoxification.
Journal ArticleDOI
Metallothionein and the Trace Minerals
Ian Bremner,John H. Beattie +1 more
TL;DR: Etude de sa degradation, de ses fonctions dans la detoxication des metaux lourds, dans le controle de l'absorption intestinale, le metabolisme hepatique du zinc and du cuivre et de proteines de transfert.
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Zinc deficiency suppresses matrix mineralization and retards osteogenesis transiently with catch-up possibly through Runx 2 modulation
In-Sook Kwun,Young-Eun Cho,Ria-Ann R. Lomeda,Hong-In Shin,Je-Yong Choi,Young-Hee Kang,John H. Beattie +6 more
TL;DR: Zhang et al. as discussed by the authors investigated whether zinc deficiency down-regulates bone marker gene transcription and whether this might occur through modulation of Runx2 and found that zinc deficiency decreased ECM mineralization in osteoblastic MC3T3-E1 cells.