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John J. Turek

Researcher at Purdue University

Publications -  167
Citations -  3506

John J. Turek is an academic researcher from Purdue University. The author has contributed to research in topics: Speckle pattern & Holography. The author has an hindex of 32, co-authored 164 publications receiving 3344 citations. Previous affiliations of John J. Turek include Indiana University & Imperial College London.

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Endocytosis of folate-protein conjugates: Ultrastructural localization in KB cells

TL;DR: The subcellular localization of protein-folic acid conjugates in KB cells was determined using folic acid-bovine serum albumin-colloidal gold (F-BSA-CG) as a tracer to demonstrate covalently conjugated proteins taken up by cells via endocytosis modify physiological processes occurring in the cytosol.
Journal Article

Indium-111-DTPA-folate as a potential folate-receptor-targeted radiopharmaceutical.

TL;DR: Indium-111-DTPA-folate appears suitable as a radiopharmaceutical for targeting tumor-associated folate receptors, and shows folate receptor-mediated uptake and retention in the kidneys, presumably reflecting radiotracer binding to folates of the proximal tubules.
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β-Catenin-regulated ALDH1A1 is a target in ovarian cancer spheroids.

TL;DR: In this paper, the role of β-catenin-regulated ALDH1A1 in the maintenance of ovarian cancer spheroids was investigated and new targetable molecules targeting this cell population were proposed.

β-Catenin-regulated ALDH1A1 is a target in ovarian cancer spheroids

TL;DR: The role of β-catenin-regulated ALDH1A1 in the maintenance of OC spheroids is supported and new ALDH 1A1 inhibitors targeting this cell population are proposed.
Journal Article

The macrophage in acute neural injury: changes in cell numbers over time and levels of cytokine production in mammalian central and peripheral nervous systems

TL;DR: The timing and density of ED-1-positive macrophage accumulation and cytokine production by macrophages in standardized compression injuries to the spinal cord and sciatic nerves of individual rats are evaluated and the meaning of these data relative to the known differences in the reparative responses of the PNS and CNS to injury is discussed.