J
John Yeh
Researcher at Harvard University
Publications - 62
Citations - 2086
John Yeh is an academic researcher from Harvard University. The author has contributed to research in topics: Medicine & Epidermal growth factor. The author has an hindex of 22, co-authored 48 publications receiving 1898 citations. Previous affiliations of John Yeh include Boston Children's Hospital & Beth Israel Deaconess Medical Center.
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Journal ArticleDOI
beta-amyloid fibrils induce tau phosphorylation and loss of microtubule binding.
TL;DR: Amyloid fibril formation alters the phosphorylation state of tau, resulting in the loss of microtubule binding capacity and somatodendritic accumulation, properties also exhibited by tau in the AD brain.
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In situ localization of apoptosis in the rat ovary during follicular atresia.
Angela Palumbo,John Yeh +1 more
TL;DR: This study represents a systematic analysis of apoptosis in the rat ovary at different functional stages and supports the hypothesis that apoptosis is involved in the process of follicular atresia.
Journal ArticleDOI
Active management of the third stage of labour with and without controlled cord traction: a randomised, controlled, non-inferiority trial
A Metin Gülmezoglu,Pisake Lumbiganon,Sihem Landoulsi,Mariana Widmer,Hany Abdel-Aleem,Mario Festin,Guillermo Carroli,Zahida Qureshi,João Paulo Souza,Eduardo Bergel,Gilda Piaggio,Shivaprasad S. Goudar,John Yeh,Deborah Armbruster,Mandisa Singata,Cristina Pelaez-Crisologo,Fernando Althabe,Peter Sekweyama,Justus Hofmeyr,Mary-Ellen Stanton,Richard J. Derman,Diana Elbourne +21 more
TL;DR: Although the hypothesis of non-inferiority was not met, omission of controlled cord traction has very little effect on the risk of severe haemorrhage and scaling up of haem orrhage prevention programmes for non-hospital settings can safely focus on use of oxytocin.
Journal ArticleDOI
β‐Amyloid Neurotoxicity in Human Cortical Culture Is Not Mediated by Excitotoxins
TL;DR: The results suggest that β‐amyloid is directly neurotoxic to primary human cortical neurons by a mechanism that does not involve excitatory amino acid receptors.
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Presence of messenger ribonucleic acid for epidermal growth factor (EGF) and EGF receptor demonstrable in monolayer cell cultures of myometria and leiomyomata.
TL;DR: In this paper, the expression of mRNAs encoding EGF and the EGF receptor in myometrial and leiomyoma cultures suggest that EGF may be involved in the autocrine/paracrine regulation of human uterine leioma and myometrium growth.