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Jun Zhou

Researcher at Agency for Science, Technology and Research

Publications -  298
Citations -  5891

Jun Zhou is an academic researcher from Agency for Science, Technology and Research. The author has contributed to research in topics: Chemistry & Computer science. The author has an hindex of 32, co-authored 200 publications receiving 3384 citations. Previous affiliations of Jun Zhou include University of Electronic Science and Technology of China & Chongqing University.

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Engineering of a Nanosized Biocatalyst for Combined Tumor Starvation and Low-Temperature Photothermal Therapy

TL;DR: A tumor-targeted redox-responsive composite biocatalyst is designed and fabricated, which may combine tumor starvation therapy and low-temperature photothermal therapy for the treatment of oxygen-deprived tumors.
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Chemically Exfoliated VSe2 Monolayers with Room-Temperature Ferromagnetism.

TL;DR: Using an electrochemical exfoliation approach with organic cations as the intercalants, monolayer 1T-VSe2 flakes are successfully obtained from the bulk crystal at high yield and density functional theory (DFT) calculations show that such effect can be minimized by physisorbed oxygen molecules or covalently bound thiol molecules.
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Hollow mesoporous silica nanoparticles facilitated drug delivery via cascade pH stimuli in tumor microenvironment for tumor therapy

TL;DR: The in vivo results demonstrated that drug loaded HMSNs significantly inhibited tumor growth while only with minimal toxic side effects, providing new insight into the development of new generation of drug delivery carriers triggering by tumor microenvironment.
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Tumor microenvironment-activatable Fe-doxorubicin preloaded amorphous CaCO3 nanoformulation triggers ferroptosis in target tumor cells.

TL;DR: An amorphous calcium carbonate (ACC)–based nanoassembly for tumor-targeted ferroptosis therapy, in which the totally degradable ACC substrate could synergize with the therapeutic interaction between doxorubicin (DOX) and Fe2+.
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Enzyme responsive mesoporous silica nanoparticles for targeted tumor therapy in vitro and in vivo

TL;DR: In vitro tests proved that the anticancer drug loading system could efficiently induce cell apoptosis in vitro, and in vivo tumor experiments confirmed that the anti-tumor loading system can efficiently inhibit tumor growth with minimal side effects.