K
Kano Hiroi
Researcher at Kyoto University
Publications - 10
Citations - 675
Kano Hiroi is an academic researcher from Kyoto University. The author has contributed to research in topics: Nitric oxide & Retina. The author has an hindex of 6, co-authored 10 publications receiving 651 citations.
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Journal ArticleDOI
Specific deficit of the ON response in visual transmission by targeted disruption of the mGluR6 gene
Masayuki Masu,Hideki Iwakabe,Yoshiaki Tagawa,Tomomitsu Miyoshi,Masayuki Yamashita,Yutaka Fukuda,Hitoshi Sasaki,Kano Hiroi,Yasuhisa Nakamura,Ryuichi Shigemoto,Masahiko Takada,Kenji Nakamura,Kazuki Nakao,Motoya Katsuki,Shigetada Nakanishi +14 more
TL;DR: The results demonstrate that mGluR6 is essential in synaptic transmission to the ON bipolar cell and that the OFF response provides an important means for transmitting visual information.
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Inducible Nitric Oxide Synthase in Retinal Ischemia-reperfusion Injury
TL;DR: This study shows that iNOS mRNA is highly expressed by non-neuronal cells of the inner retina during reperfusion following transient retinal ischemia, and shows that L-NIO treatment provides some protection against ischemIA-reperfusion injury.
Journal Article
Roles of constitutive nitric oxide synthase in postischemic rat retina.
Masanori Hangai,Kazuaki Miyamoto,Kano Hiroi,A. Tujikawa,Yuichiro Ogura,Yoshihito Honda,Nagahisa Yoshimura +6 more
TL;DR: This study shows that postischemic inhibition of NOS worsens retinal damage after ischemia-reperfusion and alters postISChemic retinal circulation.
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Effects of Intravenous Superoxide Dismutase and Catalase on Electroretinogram in the Cat Postischemic Retina
TL;DR: Results suggested that the oxygen-derived free radicals affected the retinal pigment epithelium at the early period of reperfusion.
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Role of nitric oxide during the initial phase of reperfusion after retinal ischemia in the rat.
TL;DR: Inhibition of nitric oxide synthase during the initial phase of reperfusion may worsen the recovery of the b-wave following retinal ischemia, at least in part, by inhibiting establishment of reperFusion.