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Katharine E. Hubbard

Researcher at University of Hull

Publications -  29
Citations -  2119

Katharine E. Hubbard is an academic researcher from University of Hull. The author has contributed to research in topics: Circadian rhythm & Circadian clock. The author has an hindex of 13, co-authored 24 publications receiving 1844 citations. Previous affiliations of Katharine E. Hubbard include University of Cambridge & University of California, San Diego.

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Early abscisic acid signal transduction mechanisms: newly discovered components and newly emerging questions

TL;DR: An emerging PYR/RCAR-PP2C-SnRK2 model of ABA signal transduction provides researchers with a framework through which early ABA signaling can be understood, and allow novel questions about the hormone response pathway and possible applications in stress resistance engineering of plants to be addressed.
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Photosynthetic entrainment of the Arabidopsis thaliana circadian clock

TL;DR: It is demonstrated that endogenous oscillations in sugar levels provide metabolic feedback to the circadian oscillator through the morning-expressed gene PSEUDO-RESPONSE REGULATOR 7 (PRR7), and that prr7 mutants are insensitive to the effects of sucrose on the circadian period.
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Modulation of environmental responses of plants by circadian clocks

TL;DR: How environmental rhythms of temperature and light intensity entrain the circadian clock, how photoperiodism may be regulated by the relationship between environmental rhythms and the phasing of clock outputs, and how gating modulates the sensitivity of the clock and other responses to environmental and physiological signals are described.
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The Arabidopsis circadian clock incorporates a cADPR-based feedback loop

TL;DR: It is shown that the plant circadian clock also incorporates the cytosolic signaling molecule cyclic adenosine diphosphate ribose (cADPR), which modulates the circadian oscillator's transcriptional feedback loops and drives circadian oscillations of Ca2+ release.
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How plants tell the time.

TL;DR: The advances in determining the molecular nature of the circadian oscillator are described and an architecture of several interlocking negative-feedback loops is proposed and the adaptive advantages of circadian control, with particular reference to the regulation of metabolism, are considered.