K
Keith M. Rich
Researcher at Washington University in St. Louis
Publications - 129
Citations - 5315
Keith M. Rich is an academic researcher from Washington University in St. Louis. The author has contributed to research in topics: Radiosurgery & Magnetic resonance imaging. The author has an hindex of 35, co-authored 124 publications receiving 4697 citations.
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Journal ArticleDOI
Randomized Phase II Study of Cilengitide, an Integrin-Targeting Arginine-Glycine-Aspartic Acid Peptide, in Recurrent Glioblastoma Multiforme
David A. Reardon,Karen Fink,Tom Mikkelsen,Timothy F. Cloughesy,Alison M. O'Neill,Scott R. Plotkin,Michael Glantz,Paula Ravin,Jeffrey J. Raizer,Keith M. Rich,David Schiff,William R. Shapiro,Susan Burdette-Radoux,Edward J. Dropcho,Sabine M. Wittemer,Johannes Nippgen,Martin Picard,L. Burt Nabors +17 more
TL;DR: Cilengitide monotherapy is well tolerated and exhibits modest antitumor activity among recurrent GBM patients, and additional studies integrating cilENGitide into combinatorial regimens for GBM are warranted.
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Clinical Features and Outcome in North American Adults With Moyamoya Phenomenon
Christopher L. Hallemeier,Keith M. Rich,Robert L. Grubb,Michael R. Chicoine,Christopher J. Moran,DeWitte T. Cross,Gregory J. Zipfel,Ralph G. Dacey,Colin P. Derdeyn +8 more
TL;DR: Moyamoya phenomenon in North American adults is associated with a high risk of recurrent stroke, particularly those with bilateral involvement and ischemic symptoms, and data suggest a potential benefit with surgery if diagnosis could be made earlier.
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The effects of nerve growth factor and its antiserum on the postnatal development and survival after injury of sensory neurons in rat dorsal root ganglia
TL;DR: Removal of exogenous NGF resulted in the death of the sensory neurons which had been maintained in the dorsal root ganglion of 1-day-old rats, indicating a shift toward larger sizes due to a preferential loss of small cells.
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The role of NGF in sensory neurons in vivo
TL;DR: Recent evidence is reviewed which demonstrates a broader role for NGF and for the central target in the maintenance of sensory neurons and the potential use of NGF as a pharmacological agent to ameliorate the effects of injury, and new data establishing thecentral target tissue as a source of neurotrophic support.
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Nerve growth factor enhances regeneration through silicone chambers.
TL;DR: The NGF/saline group showed a more mature-appearing regenerated nerve based on the percentage of myelinated axons, thickness of the myelin sheaths, and development of internal organization.