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Ken Katakura

Researcher at Hokkaido University

Publications -  124
Citations -  2427

Ken Katakura is an academic researcher from Hokkaido University. The author has contributed to research in topics: Cutaneous leishmaniasis & Leishmaniasis. The author has an hindex of 26, co-authored 116 publications receiving 2057 citations. Previous affiliations of Ken Katakura include Gunma University.

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Detection and identification of leishmania species within naturally infected sand flies in the andean areas of ecuador by a polymerase chain reaction

TL;DR: A polymerase chain reaction (PCR)-based method for detection of Leishmania minicircle DNA within individual sand flies, which will be a powerful tool for the rapid identification of prevalent sand fly and Leishmaniasis species as well as monitoring the infection rate in sand fly populations in endemic areas.
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Development of a loop-mediated isothermal amplification method for rapid mass-screening of sand flies for Leishmania infection

TL;DR: The robustness, superior sensitivity, and ability to produce better visual discriminatory reaction products than existing LAMP fluorescence and turbidity assays indicated the field potential usefulness of this new method for surveillance and epidemiological studies of leishmaniasis in developing countries.
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Sequence variation of the cytochrome b gene of various human infecting members of the genus Leishmania and their phylogeny.

TL;DR: The nucleotide sequences of the Cyt b gene from 13 human-infecting Leishmania species (14 strains) from the New and Old Worlds were determined, and they showed good agreement with the classification of Lainson & Shaw (1987) except for the inclusion of L. (L.) major in the L.
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Protective immunity and eosinophilia in IgE-deficient SJA/9 mice infected with Nippostrongylus brasiliensis and Trichinella spiralis.

TL;DR: The kinetics of blood eosinophilia, worm burden after primary infection, and resistance to secondary infection in SJA/9 mice were the same in both infections as those in congenic SJL/J mice used as an IgE-producing control, indicating that the host responses examined here operate under IgE