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Kendall A. Bryant

Researcher at University of Nebraska Medical Center

Publications -  5
Citations -  149

Kendall A. Bryant is an academic researcher from University of Nebraska Medical Center. The author has contributed to research in topics: Promoter & Gene mapping. The author has an hindex of 4, co-authored 5 publications receiving 143 citations.

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Journal ArticleDOI

σB and SarA independently regulate polysaccharide intercellular adhesin production in Staphylococcus epidermidis

TL;DR: It was demonstrated that PIA production could be restored in both 1457 sigB and 1457 sarA by complementing these mutants with a full-length icaADBC operon controlled by a cadmium-inducible noncognate promoter.
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KPC-4 Is Encoded within a Truncated Tn4401 in an IncL/M Plasmid, pNE1280, Isolated from Enterobacter cloacae and Serratia marcescens

TL;DR: DNA sequencing revealed that blaKPC-4 was encoded on an IncL/M plasmid, pNE1280, closely related to pCTX-M360, which was found to be a novel Tn4401 element containing a truncated tnpA and lacking tnpR, ISKpn7 left, and Tn 4401 IRL-1, which are conserved in other Tn3401 transposons.
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Assessment of Whole-Genome Mapping in a Well-Defined Outbreak of Salmonella enterica Serotype Saintpaul

TL;DR: Whole-genome mapping is a viable alternative tool for the epidemiological analysis of Salmonella food-borne disease investigations and pulsed-field gel electrophoresis was concordant with 22 of 23 isolates.
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Susceptibility of enterococci to daptomycin is dependent upon testing methodology.

TL;DR: It is suggested that MicroScan methods overestimate nonsusceptible enterococci prevalence, and the lack of correlation between methods raises questions regarding which method is most effective at confirming nonsUSceptibility.
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Genetic analysis of the Staphylococcus epidermidis Macromolecular Synthesis Operon: Serp1129 is an ATP binding protein and sigA transcription is regulated by both σA- and σB-dependent promoters

TL;DR: The transcriptional regulation of sigA by ρB provides evidence that the staphylococcal σB-dependent response is controlled at both the transcriptional and post-transcriptional level and supports the need for further investigation of its role in bacterial growth.