K
Kenneth D. Brandt
Researcher at University of Kansas
Publications - 269
Citations - 33781
Kenneth D. Brandt is an academic researcher from University of Kansas. The author has contributed to research in topics: Osteoarthritis & Cartilage. The author has an hindex of 73, co-authored 269 publications receiving 32322 citations. Previous affiliations of Kenneth D. Brandt include Indiana University & Indiana University – Purdue University Indianapolis.
Papers
More filters
Journal Article
Treatment of knee osteoarthritis: relationship of clinical features of joint inflammation to the response to a nonsteroidal antiinflammatory drug or pure analgesic.
TL;DR: In this paper, a randomized double-blind comparison of acetaminophen versus analgesic and anti-inflammatory doses of ibuprofen in the treatment of 182 subjects with knee OA was conducted.
Journal ArticleDOI
Composition and glycosaminoglycan metabolism of articular cartilage from habitually loaded and habitually unloaded sites
TL;DR: The present results suggest that the rates of GAG metabolism in loaded and unloaded cartilage under atmospheric pressure in vitro may not reflect the rates which exist in articular joints under compressive loads in vivo.
Journal ArticleDOI
Neurogenic acceleration of osteoarthrosis. The effects of previous neurectomy of the articular nerves on the development of osteoarthrosis after transection of the anterior cruciate ligament in dogs.
TL;DR: Osteoarthrotic lesions, as gauged by histological and macroscopic criteria, were more frequent and severe in dogs that had had neurectomy before transection than in those that had intact sensory nerves and an unstable joint.
Journal ArticleDOI
Triamcinolone hexacetonide protects against fibrillation and osteophyte formation following chemically induced articular cartilage damage
TL;DR: Triamcinolone hexacetonide produced a marked, dose-dependent protective effect in this model of chemically induced articular cartilage damage.
Journal ArticleDOI
Marked suppression by salicylate of the augmented proteoglycan synthesis in osteoarthritic cartilage.
TL;DR: The salicylate-induced suppression of proteogly can synthesis in the osteoarthritic cartilage was not accompanied by reversal of the defect in proteoglycan aggregation or by improvement in the (presumed) defect inroteoglycan-collagen interaction in the matrix, as reflected by the abnormally high proportion of 35S-proteoglycans present in the culture medium.