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Kevin A. Harvey

Researcher at Indiana University Health

Publications -  58
Citations -  2696

Kevin A. Harvey is an academic researcher from Indiana University Health. The author has contributed to research in topics: Endothelial stem cell & Polyunsaturated fatty acid. The author has an hindex of 28, co-authored 58 publications receiving 2550 citations. Previous affiliations of Kevin A. Harvey include Indiana University & HealthPartners.

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Journal ArticleDOI

Sphingosine 1-phosphate released from platelets during clotting accounts for the potent endothelial cell chemotactic activity of blood serum and provides a novel link between hemostasis and angiogenesis.

Denis English, +6 more
- 01 Nov 2000 - 
TL;DR: Sphingosine 1‐phosphate released from platelets during clotting accounts for the potent endothelial cell chemotactic activity of blood serum and provides a novel link between hemostasis and angiogenesis.
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Induction of endothelial cell chemotaxis by sphingosine 1-phosphate and stabilization of endothelial monolayer barrier function by lysophosphatidic acid, potential mediators of hematopoietic angiogenesis.

Denis English, +6 more
- 01 Dec 1999 - 
TL;DR: The phospholipid growth factors, PA, S1P, and LPA, display divergent and potent effects on angiogenic properties of endothelial cells andAngiogenic differentiation of endothelium cells potentially act in tandem to effectively induce neovascularization.
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Trans fatty acids and coronary heart disease.

Gary P. Zaloga, +3 more
- 01 Oct 2006 - 
TL;DR: It is hypothesized that TFAs affect membrane structure, thus altering enzymatic pathways that may subsequently induce cardiac arrhythmias and sudden death, and this work concludes that dietary TFAs from partially hydrogenated oils are a major cause of premature coronary deaths in humans.
Journal ArticleDOI

Omega-3 polyunsaturated fatty acids attenuate breast cancer growth through activation of a neutral sphingomyelinase-mediated pathway.

Min Wu, +9 more
- 10 Nov 2005 - 
TL;DR: Investigation of the effect of fish oils and their active omega‐3 fatty acid constituents on breast cancer growth and inhibition of breast cancer cell growth in culture by treatment with DHA and EPA is suggested to be mediated by activation of N‐SMYase.
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Ganoderma lucidum Suppresses Growth of Breast Cancer Cells Through the Inhibition of Akt/NF-κB Signaling

Jiahua Jiang, +4 more
- 01 Jan 2004 - 
TL;DR: The results suggest that Ganoderma lucidum inhibits the growth of MDA-MB-231 breast cancer cells by modulating Akt/NF-κB signaling and could have potential therapeutic use for the treatment of breast cancer.