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Li Ma

Researcher at Georgia Southern University

Publications -  109
Citations -  3236

Li Ma is an academic researcher from Georgia Southern University. The author has contributed to research in topics: Phosphor & Photon upconversion. The author has an hindex of 31, co-authored 100 publications receiving 2503 citations. Previous affiliations of Li Ma include University of Georgia & University of Illinois at Chicago.

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Enhanced adsorptive removal of anionic and cationic dyes from single or mixed dye solutions using MOF PCN-222

TL;DR: In this article, the authors show that zirconium-metalloporphyrin mesoMOF (PCN-222/MOF-545) exhibits excellent adsorption/removal capacities for numerous anionic and cationic dyes individually and together in solution.
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Enhanced Biological Photosynthetic Efficiency Using Light-Harvesting Engineering with Dual-Emissive Carbon Dots

TL;DR: In this paper, dual-emissive carbon dots (CDs) were used to enhance the photoabsorption of chloroplasts and intact leaves for enhanced photosynthetic properties.
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Hemorrhagic shock-induced bacterial translocation: the role of neutrophils and hydroxyl radicals.

TL;DR: Shock-induced BT and intestinal injury appear to be mediated by oxidants derived from xanthine oxidase, rather than granulocytes, and DMSO and deferoxamine appear to prevent shock- induced BT by blunting the magnitude of shock-induced mucosal injury.
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Dual-Functional Antifogging/Antimicrobial Polymer Coating.

TL;DR: The excellent antifogging behavior of the smooth coating was mainly attributed to the hydrophilic/hydrophobic balance of the partially quaternized copolymer, while the covalently bonded, hydrophobic quaternary ammonium compound rendered the coating strongly antimicrobial, as demonstrated by the total kill against both Gram-positive Staphylococcus epidermidis and Gram-negative Escherichia coli.
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Protein malnutrition predisposes to inflammatory-induced gut-origin septic states.

TL;DR: PM predisposes to mucosal damage and the development of potentially lethal gut origin septic state during periods of systemic inflammation.