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Liang Wang

Researcher at Capital Medical University

Publications -  14
Citations -  3672

Liang Wang is an academic researcher from Capital Medical University. The author has contributed to research in topics: Resting state fMRI & Supplementary motor area. The author has an hindex of 12, co-authored 12 publications receiving 3383 citations. Previous affiliations of Liang Wang include Capital University.

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Changes in hippocampal connectivity in the early stages of Alzheimer's disease: evidence from resting state fMRI.

TL;DR: It is found that functional connectivity between the right hippocampus and a set of regions was disrupted in AD, and the disrupted hippocampal connectivity to the MPFC, vACC and PCC provides further support for decreased activity in "default mode network" previously shown in AD.
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Altered functional connectivity in early Alzheimer's disease: A resting‐state fMRI study

TL;DR: The results supported previous studies that have reported an anterior–posterior disconnection phenomenon and increased within‐lobe functional connectivity in AD patients and suggest that AD may disturb the correlation/anti‐correlation effect in the two intrinsically anti‐correlated networks.
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Regional coherence changes in the early stages of Alzheimer’s disease: A combined structural and resting-state functional MRI study

TL;DR: The study found significant regional coherence decreases in the posterior cingulate cortex/precuneus (PCC/PCu) in the AD patients when compared with the normal controls and increased LFBF coherence in the bilateral cuneus, right lingual gyrus and left fusiform gyrus in theAD patients.
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Regional homogeneity changes in patients with Parkinson's disease.

TL;DR: It is demonstrated that neural activity in the resting state is changed in patients with PD, secondary to dopamine deficiency, and related to the severity of the disease.
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Changes of functional connectivity of the motor network in the resting state in Parkinson's disease.

TL;DR: It is demonstrated that the pattern of functional connectivity of the motor network in the resting state is disrupted in PD, and this change is secondary to dopamine deficiency, and related to the severity of the disease.